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10 risultati
Catatonia following biparietal infarction with spontaneous recovery.
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We describe a case of catatonic stupor following simultaneous biparietal infarction. The patient recovered, a result not previously described in catatonia caused by this pattern of cerebral infarct.
Biparietal infarctions in a patient with Catatonia.
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Catatonia, beyond a psychiatric syndrome.
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Although catatonia is a well-known psychiatric syndrome, there are many possible systemic and neurological etiologies. The aim of this case report was to present a case of a patient with cerebral venous sinus thrombosis and infarction in which catatonia was the clinical manifestation of a possible
[Takotsubo cardiomyopathy and catatonia in a patient with psychotic depression].
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Takotsubo cardiomyopathy (tcmp) is an acute, reversible disruption of the left ventricular systolic function. In many respects the clinical presentation closely resembles acute coronary syndromes (myocardial infarction). tcmp is a syndrome with a pathophysiology that is not fully understood and
Persistent catatonia for 1.5 years finally resolved with electroconvulsive therapy.
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Despite having been previously associated with schizophrenia, catatonia is more often associated with mood disorders and factors related to general medical conditions. Benzodiazepines are recommended as the first option in treatment of catatonia. For patients who do not sufficiently respond to
A case of catatonia status-post left middle cerebral artery cerebrovascular accident, treated successfully with olanzapine.
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Catatonia is a psychomotor phenomenon associated with psychiatric/medical conditions. We present a patient who developed catatonia status-post left middle cerebral artery infarct. With a Bush Francis Catatonia Rating Scale score of 43 on admission, treatment with olanzapine reduced this score to 2,
Asymmetric catalepsy after right hemisphere stroke.
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We describe the appearance of left hemineglect and striking cataleptic posturing, more prominent in left-sided extremities, in a patient without psychiatric illness. Neuroimaging demonstrated a large posterior right hemisphere infarct involving the parietal, occipital, and temporal lobes, the
Psychotic disorder and extrapyramidal symptoms associated with vitamin B12 and folate deficiency.
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Vitamin B12 and folate deficiency causing neuropsychiatric and thrombotic manifestations, such as peripheral neuropathy, subacute combined degeneration of cord, dementia, ataxia, optic atrophy, catatonia, psychosis, mood disturbances, myocardial infarction and portal vein thrombosis are well known.
Cerestat and other NMDA antagonists in ischemic stroke.
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A wealth of experimental evidence demonstrates that cerebral ischemia causes excessive release of glutamate and that glutamate contributes to ischemic injury. Glutamate antagonism by any of several mechanisms can ameliorate the extent of infarction. These antagonists comprise noncompetitive blockers
[Cotard's syndrome: Case report and a brief review of literature].
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The term "Cotard's syndrome" is used to describe a number of clinical features, mostly hypochondriac and nihilistic delusions, the most characteristic of which are the ideas "I am dead" and "my internal organs do not exist". Besides, anxious and depressed mood, delusions of damnation, possession and
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