cinnamic aldehyde/eritema

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The spectrum of contact urticaria. Wheals, erythema, and pruritus.

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A large variety of topical substances can provoke wheals of human skin within 30 minutes. The usual term for this phenomenon, "contact urticaria," is too constricting and can lead to faulty diagnosis. Sometimes, the clinical expression is limited to erythema; less often, its sole manifestation is

Release of markedly increased quantities of prostaglandin D2 from the skin in vivo in humans after the application of cinnamic aldehyde.

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BACKGROUND Cinnamic aldehyde is a common fragrance additive in foods and various health and beauty products. Application of cinnamic aldehyde to the skin of humans can induce cutaneous vasodilatation characterized by erythema, urticaria, and stinging. Previous studies have suggested that

Contact sensitivities in palmar plantar pustulosis (acropustulosis).

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Acropustulosis, or chronic palmar plantar pustulosis (PPP), is a phenomenon of recurrent sterile pustules, erythema, and scaling affecting the palms and soles. Its pathogenesis is unclear, and it is difficult to treat. The purpose of this study was to elucidate further the factors involved in

Topical non-steroidal anti-inflammatory drugs inhibit non-immunologic immediate contact reactions.

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The effects of 3 topical applications of non-steroidal anti-inflammatory drugs (NSAIDs), diclofenac 1%, naproxene 10% and salicylic acid 5% in gel vehicles, on immediate contact reactions to 500 mM benzoic acid, 500 mM cinnamic aldehyde, 50 mM methyl nicotinate, all in pet., and 14.1 M (100%)

Terfenadine does not inhibit non-immunologic contact urticaria.

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To investigate the mechanisms of non-immunologic contact urticaria (NICU), the effect of 120 mg of terfenadine (H1-antagonist) on contact reactions to methyl nicotinate, diethyl fumarate, benzoic acid, cinnamic acid, cinnamic aldehyde and dimethyl sulfoxide was studied in 20 subjects. Erythema and

The burning mouth syndrome: lack of a role for contact urticaria and contact dermatitis.

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BACKGROUND The burning mouth syndrome is poorly understood and mainly affects postmenopausal women. Dental allergens have seldom explained the burning. OBJECTIVE Eight patients (seven women, one man) (mean age 62.5 years) who wore dentures and had the burning mouth syndrome were investigated to

Acetylsalicylic acid inhibits non-immunologic contact urticaria.

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To investigate the mechanisms of non-immunologic contact urticaria (NICU), the effects of 1g + 1g of acetylsalicylic acid (ASA) on contact reactions to methyl nicotinate, diethyl fumarate, benzoic acid, cinnamic acid, cinnamic aldehyde and dimethyl sulfoxide were studied in 21 test subjects.

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