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coproporphyrin/emorragia
Il collegamento viene salvato negli appunti
9 risultati
THE EXCRETION OF COPROPORPHYRIN I FOLLOWING HEMORRHAGE IN DOGS.
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Determining zinc coproporphyrin in maternal plasma--a new method for diagnosing amniotic fluid embolism.
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We measured the concentration of zinc coproporphyrin I (ZnCP-I), a characteristic component of meconium, in maternal plasma by fluorometry after HPLC. We obtained plasma samples from 89 women: 35 at weeks 10-40 of normal pregnancy, 41 shortly after normal delivery, 4 from patients with amniotic
Biochemical diagnosis of a fatal case of Günther's disease in a newborn with hydrops foetalis.
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The birth of a male baby was induced at 32 weeks. In utero, the child presented, inter alia, signs of hydrops, hepatosplenomegaly and anaemia. Two in utero transfusions for correction of the anaemia were performed at 28 and 29 weeks, respectively. The baby rapidly presented respiratory distress with
Efficiency of leukocyte depletion filters and micro-aggregate filters following intra-operative cell salvage during cesarean delivery.
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Amniotic fluid embolism induces uterine anaphylaxis and atony following cervical laceration.
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Amniotic fluid embolism (AFE) is a rare, high-risk obstetric complication primarily found in the lungs and potentially related to anaphylaxis. Tryptase release from the mast cell reflects anaphylaxis. Case report and findings: A female, aged over 40 years, presented with uterine atony and lethal
[Prompt resuscitation by obstetric anesthesiologists saved a parturient with amniotic fluid embolism: a case report].
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Amniotic fluid embolism (AFE) is a disorder with a high mortarity rate, because it often causes sudden respiratory failure, circulatory collapse and disseminated intravascular coagulation (DIC). We present a case of AFE in which an obstetric anesthesiologist promptly initiated resuscitation of a
Erythropoietic protoporphyria terminating in liver failure.
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A patient with erythropoietic protoporphyria was observed for 28 years. At the age of 38 years, cholestatic jaundice and rapidly deteriorating liver function developed. The patient died of massive bleeding from esophageal varices. During his terminal illness, the RBC protoporphyrin level rose to
Amniotic fluid embolism: pathophysiology and new strategies for management.
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The registry program of amniotic fluid embolism (AFE) in Japan started in 2003. More than 400 hundred clinical diagnosed amniotic fluid embolism has been accumulated. Those data showed that there were two etiologies of AFE: the fetal materials create physical obstructions in the maternal
Kinetics and specificity of feline leukemia virus subgroup C receptor (FLVCR) export function and its dependence on hemopexin.
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The feline leukemia virus subgroup C receptor (FLVCR) is a heme export protein that is required for proerythroblast survival and facilitates macrophage heme iron recycling. However, its mechanism of heme export and substrate specificity are uncharacterized. Using [(55)Fe]heme and the fluorescent
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