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eburnamonine/ipoglicemia
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Role of drugs in recovery of metabolic function of rat brain following severe hypoglycemia.
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Severe hypoglycemia with isoelectric EEG induced extensive deterioration of the energy state and gross alteration of amino acid contents on the rat cerebral and cerebellar cortex. During recovery, tissue glucose concentration returned to normal, while both lactate and pyruvate concentrations
Cerebral and cerebellar metabolic changes induced by drugs during the recovery period after profound hypoglycemia.
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On rat cerebral and cerebellar cortex, severe hypoglycemia with isoelectric EEG induced extensive deterioration of the energy state and gross alteration of amino acid contents. During recovery, tissue glucose concentration returned to normal, while the rate of glycogen synthesis was slow, both
Influence of aging and exogenous substances on cerebral energy metabolism in posthypoglycemic recovery.
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In rats of different ages, acute severe hypoglycemia with isoelectric EEG induced extensive deterioration of the energy state and gross alteration of amino acid contents. During recovery of adult animals, tissue glucose concentration returned to normal, while the rate of glycogen synthesis was slow,
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