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l norepinephrine/atrofia
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Longstanding atrial fibrillation causes depletion of atrial natriuretic peptide in patients with advanced congestive heart failure.
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BACKGROUND
Congestive heart failure (CHF) is characterized by neurohormonal activation, including increased plasma concentrations of atrial natriuretic peptide (ANP) and N-terminal ANP (N-ANP). Onset of atrial fibrillation (AF) further increases these peptides, but it may be hypothesized that
Catecholamine binding to plasma membrane enriched fractions of heart and skeletal muscle.
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Binding of [3H]epinephrine to plasma membrane enriched fractions from guinea pig heart and rabbit skeletal muscle was investigated using the micropore filtration technique. [3H]Epinephrine and [3H]norepinephrine were found to be degraded rapidly in aqueous buffer at pH 7.6 and 37 degrees C.
Adenylate cyclase and catecholamine binding in plasma membrane-enriched preparations of cardiac and skeletal muscle.
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Binding of [3H] epinephrine to plasma membrane-enriched fractions from guinea pig heart and rabbit skeletal muscle was investigated using the micropore filtration technique. [3H] Epinephrine and [3H] norepinephrine were found to be degraded rapidly in aqueous buffer at pH 7.6 and 37 degrees C.
Unilateral adrenal catecholamine excess. Pheochromocytoma or possible sporadic medullary hyperplasia.
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A 60-year-old man had clinical and biochemical evidence of pheochromocytoma. Venous blood from the left adrenal gland had strikingly higher concentrations of l-norepinephrine, epinephrine, and dopamine than that from the right adrenal gland. The patient's condition was cured following left
Use of catecholamines in acute myocardial infarction.
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We review the hemodynamic effects and clinical usefulness of five natural and synthetic catecholamines. Their actions are best understood by an appreciation of the relative ability of each catecholamine to activate alpha, beta 1 and beta 2 adrenergic receptors in the myocardium and peripheral
The effect of intrastriatal injection of liposome-entrapped tyrosinase on the dopamine levels in the rat brain.
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Parkinson's disease is a neurodegenerative disorder which is mainly characterized by degeneration of the dopaminergic cells in the nigro-striatal system. Due to a lowered L-tyrosine 3-monooxygenase activity, L-tyrosine is not sufficiently transformed to L-DOPA. To date the most common therapy is the
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