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Ataxia and hypermetria caused by Parelaphostrongylus tenuis infection in llamas.
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Clinical Parelaphostrongylus tenuis infection in two captive American bison (Bison bison).
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METHODS
2 juvenile (17 and 19 months of age) male American bison (Bison bison) were examined because of acute bilateral hind limb weakness and ataxia; 1 animal also had urinary incontinence.
RESULTS
Given the clinical signs and rapid deterioration in the condition of these 2 animals, obtaining a
Eosinophilic encephalomyelitis in horses caused by protostrongylid parasites.
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Four thoroughbred horses showing lameness, ataxia, circling, depression, recumbency, and seizures, were examined. The horses had gross, pale- to dark-red manifestations and foci in the central nervous system (CNS). Multifocal to coalescing eosinophilic necrotizing encephalomyelitis was observed
Acquired cervical scoliosis attributed to Parelaphostrongylus tenuis infection in an alpaca.
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METHODS
A 2-year-old alpaca was evaluated because of acute onset of cervical scoliosis.
RESULTS
Physical examination revealed severe scoliosis of the caudal portion of the cervical vertebral column with a C-shaped curvature to the right side. No gait deficits were observed. Cervical radiography
Parelaphostrongylus tenuis in captive pronghorn antelope (Antilocapra americana) in Nebraska.
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Lesions in four captive pronghorn antelope (Antilocapra americana) naturally infected with Parelaphostrongylus tenuis in eastern Nebraska (USA) are described in this report. Animals were bright and alert with hind limb ataxia that progressed to sternal or lateral recumbency between July 28 and
Parelaphostrongylus tenuis in captive reindeer and sable antelope.
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Parelaphostrongylus tenuis caused neurologic disease in 6 reindeer (Rangifer tarandus tarandus) and 2 sable antelope (Hippotragus niger) that were housed at the National Zoological Park Conservation and Research Center in Front Royal, Va. Progressive hindlimb ataxia and weakness were seen in all
Cerebrospinal nematodosis caused by Parelaphostrongylus species in an adult bull.
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A 2-y-old Brahman bull was presented with progressive hindlimb ataxia and paraparesis that led to recumbency. Postmortem examination revealed scattered pinpoint, red-brown foci within the brainstem and gray matter of the spinal cord, and a larger lesion within the spinal cord at the level of T13.
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