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Pagina 1 a partire dal 1987 risultati
Branch atheromatous disease has a stronger association with late-onset epileptic seizures than lacunar infarction in Japanese patients.
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Late seizures following a first symptomatic brain infarct are related to large infarcts involving the posterior area around the lateral sulcus.
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Controversies exist concerning factors that contribute to the occurrence of epileptic seizures after stroke. Therefore, we studied prospectively the occurrence of seizures in 322 patients with a first-ever CT-confirmed symptomatic territorial brain infarct involving the cortex. We also studied
Catastrophic Secondary Infarctions with Onset Seizure Following Tissue Plasminogen Activator Therapy.
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Fatalities following intravenous recombinant tissue-type plasminogen activator therapy have been reported. Major fatal complications following intravenous recombinant tissue-type plasminogen activator therapy include intracranial hemorrhage, aortic dissection, and extracranial bleeding. However, the
EEG findings after a cerebral territorial infarct in patients who develop early- and late-onset seizures.
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BACKGROUND
EEG findings are generally not considered to be very helpful for the diagnosis of poststroke seizures.
OBJECTIVE
This retrospective study investigates the EEG characteristics in patients who develop seizures after a cerebral territorial infarct.
METHODS
The study population consisted of
Seizures and cerebral infarction in the full-term newborn.
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Cerebral arterial infarction is a more common cause of neonatal seizures than has been previously appreciated. In 50 full-term newborns with seizures studied, 7 had cerebral infarction which was the second most common definable cause of seizures. We describe these 7 full-term infants with
Role of associated cortical lesions in motor partial seizures and lenticulostriate infarcts.
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In a population-based study, we evaluated seizures occurring in the first 15 days after strokes among 1,640 consecutive patients who had ischemic (814 infarcts with atheroma and 126 with cardiogenic embolism, 273 lacunar infarcts, 259 transient ischemic attacks) or hemorrhagic stroke (129
Bilateral paramedian thalamic infarction initially presenting as a convulsive seizure.
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Bithalamic infarctions initially presenting as a convulsive seizure are rarely reported and, to our best knowledge, have never been reported in China. Here, we present a patient with convulsive seizure at the onset of bilateral paramedian thalamic infarction. The diffusion-weighted imaging revealed
Cerebellar Infarction: Unusual Manifestation with Facial Palsy, Focal Seizures, and Secondary Generalization.
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BACKGROUND
Cerebellar infarction is exceedingly rare in neonates, usually occurring after traumatic birth. Lifelong sequelae can result from cerebellar damage with disorders of motor function, ataxia, and also cognitive dysfunction.
OBJECTIVE
We report the clinical presentation of a preterm triplet
Protein S deficiency, epileptic seizures, sagittal sinus thrombosis and hemorrhagic infarction after ingestion of dimenhydrinate.
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Congenital protein S deficiency is associated with an increased risk of venous thrombosis. A14-year-old boy presented with epileptic seizures and thrombosis of the superior sagittal sinus and frontal hemorrhagic venous infarction after ingestion of 50 mg of dimenhydrinate, an antiemetic drug. The
[Late onset seizures in cerebral infarction--clinical study of late onset seizures in cortical branch infarction].
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We retrospectively analyzed the clinical features, prognosis, CT scans and electroencephalograms in 527 patients with cerebral infarction in the Toranomon hospital between 1983 and 1990. CT scans revealed cortical involvement in 130 cases and subcortical infarcts in the territory of the perforating
Epileptic seizures following subcortical infarcts.
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Stroke is one of the most frequent causes of seizures in adulthood. Subcortical infarcts have been associated with post-stroke epileptic seizures, although less frequently than cortical ischaemia. We studied 113 patients from a hospital stroke registry. The patients had subcortical non-lacunar
Watershed infarctions are more prone than other cortical infarcts to cause early-onset seizures.
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BACKGROUND
Early-onset seizures(ESs) have been reported in 2% to 6% of strokes. Most previous studies have been retrospective and did not systematically perform cerebral magnetic resonance imaging (MRI).
OBJECTIVE
To determine the prevalence and determinants of ESs in a prospective
Neonatal cerebral infarction: an under recognised/unreconised cause of neonatal seizures?
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Neonatal cerebral infarction or neonatal stroke is a well recognised cause of neonatal seizures. The awareness of its existence among healthcare workers is low and it is not even mentioned as a cause of neonatal seizures in most books. Many of the affected neonates are well, without any evidence of
Cerebral infarction as multifocal clonic seizures in a term neonate.
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BACKGROUND
The incidence of neonatal stroke in full-term infants has been cautiously estimated as 1:10,000, but infants can initially have few symptoms, and the condition has the potential for underdiagnosis. Follow-up studies of known full-term neonatal stroke victims beyond 3 years of age indicate
[Localized neonatal convulsions and cerebral arterial infarction].
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BACKGROUND
Stroke is a rare cause of neonatal seizures.
METHODS
During a 5-year period, eight full-term infants were admitted to hospital for seizures due to a stroke. Seizures began shortly after birth and were always one-sided. Early CT scans showed cerebral infarctions. Motor disabilities such as
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