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aspartate amino transferase/hypoxia
הקישור נשמר בלוח
10 תוצאות
Chronic magnesium deficiency decreases tolerance to hypoxia/reoxygenation injury in mouse heart.
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
OBJECTIVE
Magnesium (Mg) deficiency has been reported to be associated with the development of the metabolic syndrome, cardiovascular diseases, and sudden death. We examined the influence of chronic Mg deficiency on cardiac tolerance to hypoxia/reoxygenation injury.
METHODS
Mice were fed an
[Effects of chronic intermittent hypoxia on hepatic function and protective mechanism of adiponectin in rats].
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
Objective: To explore the effect of chronic intermittent hypoxia (CIH) on rats hepatic function, and the protective mechanism of adiponectin (Ad). Methods: Sixty healthy male wistar rats were randomly divided into 4 groups: normal control (NC), NC+ Ad, CIH, and CIH+ Ad groups with 15 rats in each.
Glycine prevents alcohol-induced liver injury by decreasing alcohol in the rat stomach.
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
OBJECTIVE
Inactivation of Kupffer cells prevents alcohol-induced liver injury, and hypoxia subsequent to a hypermetabolic state caused by activated Kupffer cells probably is involved in the mechanism. Glycine is known to prevent hepatic reperfusion injury. The purpose of this study was to determine
Murine glutathione S-transferase A1-1 in sickle transgenic mice.
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
Patients with sickle cell anemia exhibit mild to moderate renal and liver damage. Glutathione S-transferase A1-1 is produced during kidney and liver damage. We hypothesized that cellular damage in sickle transgenic mice would lead to increased serum and urine murine glutathione S-transferase A1-1
The role of amino acid catabolism in the formation of the tricarboxylic acid cycle intermediates and ammonia in anoxic rat heart.
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
Amino acid catabolism, the tricarboxylic acid cycle intermediates and ammonia formation were studied in isolated perfused rat heart under anoxia. The total net anaplerosis due to amino acid degradation in anoxia was equal to that in oxygenation (6.29 and 6.09 mumol/g dry weight per h, respectively)
[Enzymatic activity of the cerebrospinal fluid in patients with cerebral infarct].
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
Dynamic measurements in the cerebrospinal fluid (CSF) of the activities of lactate dehydrogenase (LDH), its first isoenzyme (LDH1), aspartate amino transferase (AsAT), alanine amino transferase (AlAT), creatine phosphokinase (CPK), gamma-glutamyl transpeptidase (GGT) and alkaline phosphatases (ALP)
MTMR14 protects against hepatic ischemia-reperfusion injury through interacting with AKT signaling in vivo and in vitro
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
Hepatic ischemia-reperfusion (IR) injury is characterized by severe inflammation and cell death. However, very few effective therapies are presently available for hepatic IR injury treatment. Here, we reported a protective function and the underlying mechanism of myotubularin-related protein 14
Interferon regulatory factor 9 is a key mediator of hepatic ischemia/reperfusion injury.
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
OBJECTIVE
Hepatic ischemia/reperfusion (I/R) injury is characterized by anoxic cell injury and the generation of inflammatory mediators, leading to hepatic parenchymal cell death. The activation of interferon regulatory factors (IRFs) has been implicated in hepatic I/R injury, but the role of IRF9
Exosomes derived from human umbilical cord blood mesenchymal stem cells improve hepatic ischemia reperfusion injury via delivering miR-1246.
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
The purpose of this study was to explore the associated mechanism by which MSCs-derived exosomes exerted protective effect in hepatic ischemia/reperfusion injury (IRI). Human umbilical cord blood mesenchymal stem cells (hUCB-MSCs)-derived exosomes were administrated into LO2 cells exposed to
Alda-1, an ALDH2 activator, protects against hepatic ischemia/reperfusion injury in rats via inhibition of oxidative stress.
רק משתמשים רשומים יכולים לתרגם מאמרים
התחבר הרשם
Previous studies have proved that activation of aldehyde dehydrogenase two (ALDH2) can attenuate oxidative stress through clearance of cytotoxic aldehydes, and can protect against cardiac, cerebral, and lung ischemia/reperfusion (I/R) injuries. In this study, we investigated the effects of the ALDH2
המאגר השלם ביותר של צמחי מרפא המגובה על ידי המדע
- עובד ב 55 שפות
- מרפא צמחי מרפא מגובה על ידי מדע
- זיהוי עשבי תיבול על ידי דימוי
- מפת GPS אינטראקטיבית - תייגו עשבי תיבול במיקום (בקרוב)
- קרא פרסומים מדעיים הקשורים לחיפוש שלך
- חפש עשבי מרפא על פי השפעותיהם
- ארגן את תחומי העניין שלך והתעדכן במחקר החדשות, הניסויים הקליניים והפטנטים
הקלד סימפטום או מחלה וקרא על צמחי מרפא שעשויים לעזור, הקלד עשב וראה מחלות ותסמינים שהוא משמש נגד.
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