beta hydroxybutyric acid/vėmimas

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[Diabetic ketoacidosis masked by metabolic alkalosis].

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Diabetic ketoacidosis is caused by accumulation of acetoacetic and β-hydroxybutyric acids and is a common acute metabolic complication of type 1 diabetes mellitus. A much less common and recognized metabolic complication is diabetic ketoacidosis with alkalaemia ("ketoalkalosis"). Diuretic use,

Case of alcoholic ketoacidosis accompanied with severe hypoglycemia.

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We report a 55 year old Japanese man with a history of alcohol abuse, who was in a near fasting state for the previous few days.He was admitted to our hospital with abrupt disturbance of consciousness. He presented disturbance of consciousness with extreme hypoglycemia and ketoacidosis with high

[Prospective multicenter study on long-term ketogenic diet therapy for intractable childhood epilepsy].

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OBJECTIVE To evaluate the efficacy and safety of long-term ketogenic diet (KD) on the children with intractable epilepsy. METHODS This was a prospective, open-label study of intractable epilepsy patients treated with the classic KD with a lipid-to-nonlipid ratio 4:1 between October 2004 and July

High anion gap metabolic acidosis induced by cumulation of ketones, L- and D-lactate, 5-oxoproline and acute renal failure.

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BACKGROUND Frequent causes of high anion gap metabolic acidosis (HAGMA) are lactic acidosis, ketoacidosis and impaired renal function. In this case report, a HAGMA caused by ketones, L- and D-lactate, acute renal failure as well as 5-oxoproline is discussed. METHODS A 69-year-old woman was admitted

Ketoacidosis associated with ketogenic diet in a non-diabetic lactating woman

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A 37-year-old woman who had 8 weeks post partum, breast feeding and on a low carbohydrate and high protein (ketogenic) diet, was admitted to the hospital with acute onset of nausea, vomiting and abdominal pain of 1-day duration. On admission, she was found to have high anion gap metabolic acidosis,

Metabolic acidosis in the alcoholic: a pathophysiologic approach.

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The purpose of this paper is to review the acid-base abnormalities in patients presenting with metabolic acidosis due to acute ethanol ingestion and to review the theoretical constraints on ethanol metabolism in the liver. Alcohol-induced acidosis is a mixed acid-base disturbance. Metabolic acidosis

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