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10 rezultatus
[The effect of 17beta-estradiol and estriol on experimental dietetic liver necrosis in the rat].
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[DEGENERATION AND NECROSIS OF THE LIVER AND KIDNEY IN RATS AFTER THE ADMINISTRATION OF HIGH DOSES OF 17-BETA-ESTRADIOL AND ESTRIOL].
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Estriol sensitizes rat Kupffer cells via gut-derived endotoxin.
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The relationship between gender and alcohol-induced liver disease is complex; however, endotoxin is most likely involved. Recently, it was reported that estriol activated Kupffer cells by upregulation of the endotoxin receptor CD14. Therefore, the purpose of this work was to study how estriol
Likelihood ratios for the prediction of preterm delivery with biomarkers.
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OBJECTIVE
To conduct a literature search for selected biomarkers on preterm delivery and estimate their likelihood ratios (LR).
METHODS
Structured review.
METHODS
Low and high-risk populations and women with symptoms of preterm delivery. METHODS. Publications were identified in PubMed.
METHODS
LR on
Estrogens and cell death in murine uterine luminal epithelium.
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The luminal epithelium of adult ovariectomized mice responds to estradiol-17 beta with a synchronised wave of DNA synthesis and mitosis. Estriol, however, although producing a similar DNA-synthetic and mitotic response fails to cause an increase in cell number owing to a wave of cell death occurring
Combined histochemical and biochemical analysis of sex hormone dependence of ferric nitrilotriacetate-induced renal lipid peroxidation in ddY mice.
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Ferric nitrilotriacetate (Fe-NTA) induces renal proximal tubular necrosis, a consequence of lipid peroxidation, that finally leads to a high incidence of renal adenocarcinoma in rats and mice. Male animals are much more susceptible than female animals to both effects. Moreover, the distribution of
Oral contraceptives worsen endotoxin-induced liver injury in rats.
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BACKGROUND
Oral contraceptives are widely used; however, these drugs occasionally cause liver injury. Recently, it was reported that estriol worsens alcoholic liver injury by the mechanism involving activation of Kupffer cells as a result of gut-derived endotoxin. However, the relationship between
The effects of compounded bioidentical transdermal hormone therapy on hemostatic, inflammatory, immune factors; cardiovascular biomarkers; quality-of-life measures; and health outcomes in perimenopausal and postmenopausal women.
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Menopause impacts 25 million women world wide each year, and the World Health Organization estimates 1.2 billion women will be postmenopausal by 2030. Menopause has been associated with symptoms of hot flashes, night sweats, dysphoric mood, sleep disturbance, and conditions of cardiovascular
Inonotus obliquus polysaccharide protects against adverse pregnancy caused by Toxoplasma gondii infection through regulating Th17/Treg balance via TLR4/NF-κB pathway.
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The study aimed to explore the protective effects of Inonotus obliquus polysaccharide (IOP) on adverse pregnancy caused by Toxoplasma gondii (T. gondii) infection and discover its potential mechanisms in pregnant mice. Our data showed that IOP significantly reduced the abortion rate, inhibited the
Estrogenic endocrine-disrupting chemicals modulate the production of inflammatory mediators and cell viability of lipopolysaccharide-stimulated macrophages.
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Estrogenic endocrine-disrupting chemicals (EDCs) are exogenous substances that act as competitive inhibitors of estrogen in the endocrine system. By disrupting the endocrine system, EDCs can cause severe disabilities and diseases, including cancers and altered sexual development. Although the
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