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9 rezultatus
Thymosin beta 4 promotes corneal wound healing and decreases inflammation in vivo following alkali injury.
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Prisijungti Registracija
Previously, thymosin beta 4 (Tbeta(4)) was found to promote wound healing in full thickness skin wounds and heptanol debrided corneas. Here, the effect of Tbeta(4) was examined treatment on corneal wound healing and inflammation in vivo after alkali injury, a more severe wound of the eye. Corneas
Epithelial abrasion precipitates stromal ulceration in the vitamin A--deficient rat cornea.
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Although the role of vitamin A deficiency in the development of xerophthalmia is well established, there is still some question as to whether the deficiency alone is sufficient cause for the development of keratomalacia. This article describes the clinical, histologic, and microbiologic changes
Gap junctional communication between murine macrophages and intestinal epithelial cell lines.
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In intestinal inflammation, inflammatory cells infiltrate the submucosa and are found juxtaposed to intestinal epithelial cell (IEC) basolateral membranes and may directly regulate IEC function. In this study we determined whether macrophage (M phi), P388D1 and J774A.1, are coupled by gap junctions
[Corneal burns and matrix metalloproteinases (MMP-2 and -9): the effects of human amniotic membrane transplantation].
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OBJECTIVE
Human amniotic membranes have recently been used in ophthalmology to restore deleted ocular surface after burns. Matrix metalloproteinases-2 and -9 have been implicated in the development of neovascularization. In this study, MMP-2 and MMP-9 expression was analyzed by in situ zymography on
Reconstruction of ocular surface with heterologous limbal epithelium and amniotic membrane in a rabbit model.
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OBJECTIVE
To report in vivo reconstruction of the ocular surface using amniotic membrane and heterologous transplants of epithelial limbal cells in rabbits with chemical burns.
METHODS
After severe damage to the ocular surface with n-heptanol and keratectomy, 15 rabbits developed total limbal
The effects on inhibition of corneal neovascularization after human amniotic membrane transplantation in severely damaged rabbit corneas.
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Human amniotic membrane isolated from the placenta contained basement membrane components such as type IV collagen, laminin, and 6 and 4 integrins, all of which remained detectable while preserved in glycerin for one week. One month after the n-heptanol removal of the total corneal epithelium and
Inhibition of gap junction composed of Cx43 prevents against acute kidney injury following liver transplantation.
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Postoperative acute kidney injury (AKI) is a severe complication after liver transplantation (LT). Its deterioration and magnification lead to the increase in mortality. Connexin43 (Cx43) mediates direct transmission of intracellular signals between neighboring cells, always considered to be the
Transplantation of preserved human amniotic membrane for surface reconstruction in severely damaged rabbit corneas.
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After n-heptanol removal of the total corneal epithelium and a limbal lamellar keratectomy, 23 rabbit eyes developed features of limbal stem cell deficiency including conjunctival epithelial ingrowth, vascularization and chronic inflammation. One month later, 10 control eyes received a total
A novel signaling mechanism between gas and blood compartments of the lung.
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Propagation of inflammatory signals from the airspace to the vascular space is pivotal in lung inflammation, but mechanisms of intercompartmental signaling are not understood. To define signaling mechanisms, we microinfused single alveoli of blood-perfused rat lung with TNF-alpha, and determined in
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