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[Isolation and pharmacological properties of analgesic fraction from venom of Naja Naja atra].

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Ielogoties Reģistrēties

OBJECTIVE To separate main analgesic fraction from venom of Guangdong Naja naja atra, to establish the basis for the using of Naja naja atra and find new analgesic fraction. METHODS Affinity chromatography and size exclusion were used to isolate the analgesic fraction from the venom of Naja naja

Peripheral and spinal antihyperalgesic activity of najanalgesin isolated from Naja naja atra in a rat experimental model of neuropathic pain.

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Ielogoties Reģistrēties

Snake venoms are a rich source of various compounds that have applications in medicine and biochemistry. Recently, it has been demonstrated that najanalgesin isolated from the venom of Naja naja atra exerts analgesic effects on acute pain in mice. The objective of this study was to evaluate the

Purification and characterization of a novel antinociceptive toxin from Cobra venom (Naja naja atra).

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Ielogoties Reģistrēties

Snake venoms have demonstrated antinociceptive activity, and certain isolated neurotoxins have demonstrated significant analgesia in animal models. Here we report a novel analgesic toxin which was isolated from Naja naja atra and was given the name 'najanalgesin'. The LD(50) of the crude venom and

Suppression of Inflammation and Arthritis by Orally Administrated Cardiotoxin from Naja naja atra.

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Ielogoties Reģistrēties

Cardiotoxin (CTX) from Naja naja atra venom (NNAV) reportedly had analgesic effect in animal models but its role in inflammation and arthritis was unknown. In this study, we investigated the analgesic, anti-inflammatory, and antiarthritic actions of orally administered CTX-IV isolated from NNAV on

[Influence of najanalgesin from Naja naja on GLT-1 in spinal cord of rat in neuropathic pain].

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Ielogoties Reģistrēties

OBJECTIVE To investigate the influence of najanalgesin on glutamate-glial transporter 1(GLT-1) in spinal cord of rats after L5 spinal nerve ligation and transection (SNL), and explore the spinal analgesic mechanism of najanalgesin. METHODS One hundred male SD rats were randomly divided into 6

Anti-inflammatory effects of Neurotoxin-Nna, a peptide separated from the venom of Naja naja atra.

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Ielogoties Reģistrēties

BACKGROUND Neurotoxin-Nna (NT), an analgesic peptide separated from the venom of Naja naja atra, has reported to have an exceptional specificity to block transmission of the nerve impulse by binding to the α- subunit of the nicotinic acetylcholine receptor in the membrane. However, little

Antinociceptive Effect of Najanalgesin from Naja Naja Atra in a Neuropathic Pain Model via Inhibition of c-Jun NH2-terminal Kinase.

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Ielogoties Reģistrēties

BACKGROUND Najanalgesin, a toxin isolated from the venom of Naja naja atra, has been shown to exert significant analgesic effects in a neuropathic pain model in rats. However, the molecular mechanism underlying this protective effect of najanalgesin is poorly understood. The present study sought to

A short-chain alpha-neurotoxin from Naja naja atra produces potent cholinergic-dependent analgesia.

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Ielogoties Reģistrēties

Objective To investigate the analgesia induced by cobrotoxin (CT) from venom of Naja naja atra, and the effects of atropine and naloxone on the antinociceptive activity of CT in rodent pain models. Methods CT was administered intraperitoneally (33.3, 50, 75 mu g/kg), intra-cerebral venticularly (2.4

Anti-Inflammatory and Immune Regulatory Actions of Naja naja atra Venom.

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Ielogoties Reģistrēties

Naja naja atra venom (NNAV) is composed of various proteins, peptides, and enzymes with different biological and pharmacological functions. A number of previous studies have reported that NNAV exerts potent analgesic effects on various animal models of pain. The clinical studies using whole venom or

Naja atra venom peptide reduces pain by selectively blocking the voltage-gated sodium channel Nav1.8.

Rakstu tulkošanu var veikt tikai reģistrēti lietotāji

Ielogoties Reģistrēties

The voltage-gated sodium channel Nav1.8 is preferentially expressed in peripheral nociceptive neurons and contributes to inflammatory and neuropathic pain. Therefore, Nav1.8 has emerged as one of the most promising analgesic targets for pain relief. Using large-scale screening of various

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