Lappuse 1 no 585 rezultātiem
We report a case of unilateral pulmonary edema due to the decompensation of an asymptomatic ipsilateral pulmonary venous stenosis by a contralateral pulmonary embolism. Emergency surgery included pulmonary embolectomy and refashioning of the stenotic pulmonary venous anastomosis.
We report the unique occurrence of bilateral pulmonary edema in a patient with a small focal pulmonary embolus without evidence of underlying cardiac or pulmonary disease. The most likely mechanism for this involves the release of humoral factors leading to extravasation of fluid across pulmonary
We report the case of a patient with a febrile acute respiratory failure associated with alveolar opacities localized in the left upper lobe on chest-X-ray. Diagnosis was related to pulmonary embolism with overflow pulmonary edema. Complete recovery was obtained after mechanical ventilation,
Thirty six patients that died with pulmonary thromboembolism (PTE) were retrospectively followed up, whose diagnosis had been made both by the classical instrumental investigations and by angiopulmography and scintigraphy of the lungs. The diagnosis pulmonary edema in PTE was made on the base of the
We report here the occurrence of focal pulmonary edema within 4 h after massive acute pulmonary embolism. The edema appeared to develop only in areas with intact pulmonary arterial blood flow and occurred in the apparent absence of left ventricular dysfunction. This pattern of pulmonary edema after
High-altitude pulmonary edema (HAPE) is a form of noncardiogenic pulmonary edema. The pathophysiology of HAPE remains unclear. A case of HAPE was associated with pulmonary thromboembolism of a left upper pulmonary artery. Pulmonary thromboembolism was an important factor in development of HAPE in
High-altitude pulmonary edema (HAPE) is a recognized risk of rapid ascent to high altitude. Since the recognition of this entity more than 30 years ago, most pulmonary deaths at high altitude have been attributed to HAPE. However, as the bodies can almost never be recovered for postmortem
Pandey, Prativa, Benu Lohani, and Holly Murphy. Pulmonary embolism masquerading as high altitude pulmonary edema at high altitude. High Alt Med Biol. 17:353-358, 2016.-Pulmonary embolism (PE) at high altitude is a rare entity that can masquerade as or occur in conjunction with high altitude
We report here the occurrence of acute focal pulmonary edema after thrombolytic therapy for massive pulmonary embolism. Symptomatic pulmonary edema developed in a 75-yr-old man after streptokinase infusion for a massive pulmonary embolism. Repeat radiographic studies demonstrated that the edema
A case of immunologic pulmonary edema secondary to hydrochlorothiazide allergy developed in a 55-year-old woman that clinically simulated pulmonary embolism. The patient had abnormal washin images with normal washout images on an Xe-133 ventilation study. On the perfusion study, large bilateral
Although hypersensitivity reactions secondary to recombinant tissue plasminogen activator (rtPA) are rarely encountered, they may have important consequences. In this case presentation, oropharyngeal angioneurotic edema due to rtPA following pulmonary thromboembolism is presented. On the 4th hour of
A young, pregnant woman with angiographically proved pulmonary emboli developed pulmonary edema and wheezing without evidence of left ventricular failure. This cast study points out the unusual association of pulmonary embolism with pulmonary edema, wheezing, and hyper-reactive airways in a patient
OBJECTIVE
To differentiate the high altitude pulmonary edema (HAPE) from pulmonary embolism (PE) by clinical probability model of PE, lactate dehydrogenase (LDH), aspartate transaminase (AST) and D-dimer assays at high altitude.
METHODS
A prospective study.
METHODS
The study was carried out at CMH,
We report a case of severe pulmonary embolism in a 37 years old man admitted to the intensive care unit for severe acute respiratory failure. The presenting signs and symptoms were typical for severe pulmonary oedema. Chest radiograph shortly after admission showed local alveolar shadows. In the
Thirty six decreased with pulmonary thrombembolism (PTE) were analytically studied. 18 of them being with concomitant pulmonary edema (PE), confirmed pathologoanatomically. The incidence established was in 50 per cent of all the deceased, most often in combination with the unfavourable--factors