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compartment syndromes/glutathione
Врската е зачувана во таблата со исечоци
11 резултати
The role of oxygen-free radical in the apoptosis of enterocytes and bacterial translocation in abdominal compartment syndrome.
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BACKGROUND
The purpose of this study was to study the impact of intra-abdominal hypertension (IAH) on the intestine.
METHODS
One hundred and twenty Sprague-Daley rats were divided into four groups. In the ACS group, the intra-abdominal pressure (IAP) was increased to 20 mmHg. In the ACS/DE group,
Biochemical Changes in Experimental Rat Model of Abdominal Compartment Syndrome.
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BACKGROUND
Increased intra-abdominal pressure (IAP) causes tissue ischemia, subsequent hypoxia, and impairment of normal tissue metabolism. Elevation of IAP above 20 mmHg leads to progression of abdominal compartment syndrome (ACS) that is associated with organ dysfunction or failure not previously
The preventive effect of dopamine infusion in rats with abdominal compartment syndrome.
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BACKGROUND
The most significant perfusion disorder of the intra-abdominal viscera occurs in the abdominal compartment syndrome (ACS). Free oxygen radicals diffuse into the body during the reperfusion phase of ACS. Our aim was to determine the effects of dopamine infusion (3 μg/kg/min) on renal
The effect of glutamine on oxidative damage in an experimental abdominal compartment syndrome model in rats.
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BACKGROUND
The aim was to investigate whether or not glutamine, an antioxidant effective amino acid, improves the reperfusion-induced oxidative injury of abdominal hypertension.
METHODS
Wistar Albino rats were used. Group 1: Abdominal compartment syndrome alone: With the rats under anesthesia,
Melatonin ameliorates oxidative organ damage induced by acute intra-abdominal compartment syndrome in rats.
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Acutely increased intra-abdominal pressure (IAP) can lead to multiple organ failure. As blood flow to intra-abdominal organs is reduced by high venous resistance, ischemia-reperfusion (I/R) injury plays an important role in the pathogenesis of abdominal compartment syndrome (ACS) following IAP.
Hypoxic renal injury in newborns with abdominal compartment syndrome (clinical and experimental study).
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BackgroundSurgical treatment for gastroschisis and congenital diaphragmatic hernia (CDH) commonly leads to abdominal compartment syndrome (ACS) associated with hypoxic renal injury. We hypothesized that measurement of urinary and serum concentrations of vascular endothelial growth factor (VEGF),
[Protective effects of melatonin on ischemia-reperfusion injury of skeletal muscle].
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OBJECTIVE
In this study, we investigated the antioxidant protective effects of melatonin on skeletal muscles of Wistar albino-type rats with acute ischemia/reperfusion (I/R) injury.
METHODS
Twenty-eight Wistar albino-type male rats weighing between 334 to 422 g were included in this experimental
Oxytocin protects rat skeletal muscle against ischemia/reperfusion injury.
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BACKGROUND
Oxytocin (OXY) is a well-known nonapeptide that functions in reproduction. It is also known as an antioxidant in several organs. However, little is about its role in the protection of tissue against ischemia/reperfusion injury in skeletal muscle. The aim of this study was to evaluate the
Octreotide improves reperfusion-induced oxidative injury in acute abdominal hypertension in rats.
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Ischemia/reperfusion injury plays an important role in the pathogenesis of abdominal compartment syndrome, which is characterized by increased intra-abdominal pressure. The aim of this study was to investigate whether octreotide, a synthetic somatostatin analogue, improves the reperfusion injury
Octreotide: a new approach to the management of acute abdominal hypertension.
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Acutely increased intra-abdominal pressure (IAP) may lead to abdominal compartment syndrome (ACS), which ischaemia/reperfusion (I/R) injury plays an important role. The main goal of the management of ACS is to lower the intra-abdominal pressure despite reperfusion injury. Octreotide (OCT), a
Melatonin protects against ischemia/reperfusion injury in skeletal muscle.
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Melatonin has been shown to diminish ischemia-reperfusion (I/R) injury in many tissues. The main aim of this study was to evaluate the protective antioxidant effect of melatonin in skeletal muscle during I/R injury. Wistar albino rats were randomly divided into three groups. Hindlimb ischemia was
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