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dysbiosis/carbohydrate

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Страница 1 од 193 резултати

Impaired carbohydrate digestion and transport and mucosal dysbiosis in the intestines of children with autism and gastrointestinal disturbances.

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Gastrointestinal disturbances are commonly reported in children with autism, complicate clinical management, and may contribute to behavioral impairment. Reports of deficiencies in disaccharidase enzymatic activity and of beneficial responses to probiotic and dietary therapies led us to survey gene

[Carbohydrate adaptive substance for prophylaxis of immune disorders and correction of dysbacterioses].

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Effects of a new preparation, adaptokhit, on the intestinal microbiocenosis and some parameters of immune resistance were studied in Macaque rhesus during 13-day hypokinesia. Experimental animals were given adaptokhit as an alimentary supplement preventing dysbiotic consequences of the microecologic

Dietary Carbohydrate Constituents Related to Gut Dysbiosis and Health.

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Recent studies report that microbiota in the human intestine play an important role in host health and that both long- and short-term diets influence gut microbiota. These findings have fueled interest in the potential of food to promote health by shaping the intestinal microbiota. Despite the fact

Interleukin-37 sensitize the elderly type 2 diabetic patients to insulin therapy through suppressing the gut microbiota dysbiosis.

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The morbidity and prevalence of type 2 diabetes mellitus (DM) are increasing in the elderly population. Interleukin 37 (IL-37) play important roles in anti-inflammatory and anti-bacteria immune responses, but its role in the development of type 2 DM in the elderly is unclear.

Gastrointestinal dysbiosis following diethylhexyl phthalate exposure in zebrafish (Danio rerio): Altered microbial diversity, functionality, and network connectivity

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Microbiome community structure is intimately involved in key biological functions in the gastrointestinal (GI) system including nutrient absorption and lipid metabolism. Recent evidence suggests that disruption of the GI microbiome is a contributing factor to metabolic disorders and obesity. Poor

Bacillus amyloliquefaciens Ameliorates Dextran Sulfate Sodium-Induced Colitis by Improving Gut Microbial Dysbiosis in Mice Model.

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Several Bacillus strains exert beneficial effects on the maintenance of intestinal homeostasis and host health. However, whether Bacillus amyloliquefaciens (BA) can improve gut microbial dysbiosis and ameliorate colitis is unknown. Therefore, we conducted the present study to

Dietary Changes and Gut Dysbiosis in Children With Type 1 Diabetes.

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OBJECTIVE Gut dysbiosis in type 1 diabetes (T1D), characterized by high Bacteroides proportion, tends to reverse as T1D progresses, without reaching full recovery. Since diet influences microbiota structure, the aim was to evaluate the impact of dietary changes on Bacteroides proportion the first

[Influence of enterococci on the recovery of intestinal microflora changed by antibiotic therapy and carbohydrate nutrition].

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The process of intestinal microflora normalization after a course of antibiotic therapy was studied on mice and in persons with using S. faecium UDS-86. It was shown that oral inoculation of strain UDS-86 influenced correction of the intestinal microflora in the mice and persons after the antibiotic

Potential interest of gut microbial changes induced by non-digestible carbohydrates of wheat in the management of obesity and related disorders.

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OBJECTIVE The modulation of the gut microbiota by nutrients is of interest in order to reverse host metabolic alterations linked to gut microbiota dysbiosis. This review discusses how nondigestible carbohydrates (NDC) derived from wheat may constitute functional cereal food products in the

Clinical applications of urinary organic acids. Part 2. Dysbiosis markers.

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Part 1 of this series focused on urinary organic acids as markers of detoxification; part 2 focuses on dysbiosis markers. Intestinal microbial growth is accompanied by the release of products of their metabolism that may be absorbed and excreted in urine. Several organic acids are known to be

Gut microbiota dysbiosis associated with glucose metabolism disorders and the metabolic syndrome in older adults.

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Obesity and associated metabolic disorders have become highly prevalent diseases worldwide, and the human gut microbiota, due to its influence on host energy metabolism, has been attributed an important role therein. This pilot study explores host-microbiota relationships in men and women affected

High-throughput sequencing provides insights into oral microbiota dysbiosis in association with inflammatory bowel disease

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Although the prevalence of inflammatory bowel disease (IBD) has been increasing worldwide, the etiology remains elusive. Investigating oral microbiota dysbiosis is essential to understanding IBD pathogenesis. Our study evaluated variations in salivary microbiota and identified potential associations

The GC-MS metabolomics signature in patients with fibromyalgia syndrome directs to dysbiosis as an aspect contributing factor of FMS pathophysiology.

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Fibromyalgia syndrome (FMS) is a chronic pain syndrome. Previous analyses of untargeted metabolomics data indicated altered metabolic profile in FMS patients.We report a semi-targeted explorative metabolomics study on the urinary metabolite profile of FMS

Altered tissue metabolites correlate with microbial dysbiosis in colorectal adenomas.

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Several studies have linked bacterial dysbiosis with elevated risk of colorectal adenomas and cancer. However, the functional implications of gut dysbiosis remain unclear. Gut bacteria contribute to nutrient metabolism and produce small molecules termed the "metabolome", which may contribute to the

Intestinal dysbiosis and probiotic applications in autoimmune diseases.

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In humans, a complex interaction between the host immune system and commensal microbiota is required to maintain gut homeostasis. In this symbiotic relationship, the microbiota provides carbohydrate fermentation and digestion, vitamin synthesis and gut-associated lymphoid tissue development, as well
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