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Страница 1 од 48 резултати
[Effect of fructose-1,6-diphosphate and dexamethasone on ischemia/reperfusion injury after hemorrhagic shock in rabbits].
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OBJECTIVE
To investigate the mechanism of myocardial ischemia/reperfusion injury and the protective effect of fructose-1, 6-diphosphagic (FDP) and dexamethasone (DXM) in hemorrhagic shock in rabbits.
METHODS
Using a hemorrhagic shock model of Wiggers, 48 rabbits were randomly divided into 6 groups.
Haemorrhagic diathesis as a possible early sign of hereditary fructose intolerance.
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An infant girl three weeks of age with the leading symptom of skin haemorrhages is presented. On further investigation, the signs of severe hepatic damage with hypofibrinogenaemia and prothrombin complex impairment, and renal tubular dysfunction were disclosed. All these pathological symptoms, which
Increasing survival of dogs subjected to hemorrhagic shock by administration of fructose 1-6 diphosphate.
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Previous reports from this laboratory described animal experiments in which intravenous administration of fructose 1-6 diphosphate (FDP) at the onset of hypovolemia, toxemia, and trauma effected improvement in hemodynamic and metabolic parameters, attenuation of tissue damage, and a significant
Successful treatment of irreversible hemorrhagic shock in dogs with fructose-1,6 diphosphate and dichloroacetate.
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Hemodynamic and metabolic effects of fructose-1,6-diphosphate (F.D.P.) and dichloroacetate sodium (D.C.A.) administration were studied in 17 mongrel dogs during experimentally induced hemorrhagic shock using a modified Wigger's technique. During the oligemic period, which was maintained for 3 hours,
[The effect of intravenous glucose load on levels of glucose and fructose and activity of glucose-6-phosphate isomerase in serum of patients with subarachnoid hemorrhage].
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The purpose of the study was to evaluate the effect of glucose load on changes of glucose and fructose concentrations in the blood and phosphohexose isomerase activity (PHI) in the serum of patients with subarachnoid haemorrhage. The investigation was applied to 20 patients with subarachnoid
Alterations in hepatic 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatase and glucose-6-phosphatase gene expression after hemorrhagic hypotension and resuscitation.
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The mRNA abundance of several hepatic glycolytic and gluconeogenic enzymes and blood hormone concentrations were determined in hemorrhagic hypotension-induced rats before and after resuscitation with lactated Ringer's. Northern blot analysis of total liver RNA after 30 min of hemorrhage showed
Beneficial effects of fructose 1,6-diphosphate on hemorrhagic shock in rats.
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OBJECTIVE: To investigate the effects of fructose 1,6-diphosphate (FDP) on experimental hemorrhagic shock in rats. METHODS: Sixty rats were randomly divided into three groups: the normal saline control group (group A), the 5% glucose solution control group (group B) and the 5% FDP solution treated
Gastroprotective effects of honey and glucose-fructose-sucrose-maltose mixture against ethanol-, indomethacin-, and acidified aspirin-induced lesions in the rat.
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The gastric cytoprotective properties of natural honey (monofloral and polyfloral specimens) and of a glucose-fructose-sucrose-maltose mixture (GFSM) was evaluated in the rat using absolute ethanol, indomethacin and acidified acetylsalicylic acid (ASA-HCl) as necrotising agents. Prior gastric
[Effect of intravenous glucose load on blood glucose and fructose level and glucose-6-phosphate isomerase activity in serum of patients with acute cerebrovascular disease in its earliest phase].
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The purpose of the study was to investigate the dynamics of glucose and fructose concentrations in the blood changes and phosphohexose isomerase activity (PHI) in serum of unfed or after intravenous glucose injection patients, with acute cerebrovascular disease in the earliest period of illness. The
Fructose-1,6-diphosphate, when given five minutes after injury, does not ameliorate hypoxic ischemic injury to the central nervous system in the newborn pig.
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Hypoxic ischemic injury to the brain was induced in 12 0- to 3-day-old piglets. At time 0, the carotid arteries were ligated, and the blood pressure was reduced by one third by hemorrhage. At 15 min, inspired FIO2 was reduced from 50 to 6%. After 10 min of flat EEG, the FIO2 was changes to 100%, the
The mechanisms of refilling of xylem conduits and bleeding of tall birch during spring.
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Seasonal variations in osmolality and components of xylem sap in tall birch trees were determined using several techniques. Xylem sap was extracted from branch and trunk sections of 58 trees using the very rapid gas bubble-based jet-discharge method. The 5-cm long wood pieces were taken at short
Effects of hyperosmolality on the central nervous system and intracranial hemorrhage.
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The relationship between hyperosmolality and intracranial hemorrhage as well as brain dysfunction was studied. Hyperosmolality was induced by continuous infusion of hyperosmotic solutions, such as 7% sodium bicarbonate (1,666 mOsm/1), 4.64% sodium chloride (1,666 mOsm/1), 20% glucose (about 1,200
[The negative effect of enkephalins on kidney energetics in rats with hemorrhages].
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Content of adenine nucleotides, lactic and pyruvic acids, glucose and fructose-6-phosphate was studied in kidney of rats with hemorrhage constituting 3% of body mass after intravenous administration at a dose of 100 mg/kg of agonists of mu-opiate receptors DAGO and delta-opiate receptors DADL and
Effect of fructose-1, 6-diphosphate versus diphenhydramine on mortality in compound 48/80-induced shock.
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Fructose-1,6-diphosphate (FDP) has a salutary effect on hemorrhagic, traumatic and endotoxic shock. The role of FDP on compound 48/80-induced shock was therefore investigated. Sprague Dawley aged male rats (448+/-7.4 gm body weight) were randomly assigned into three groups and treated
Hemodynamics and metabolic effects of fructose 1-6 diphosphate in ischemia and shock--experimental and clinical observations.
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Numerous interventions have been used to protect organ systems and cellular viability from the lethal injury accompanying hypoperfusion and ischemia. Some measures have been directed toward improving perfusion, while others have attempted to enhance the metabolic processes. Our work has focused for
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