adrenaline/beroerte

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Effects of depleting central and peripheral adrenaline stores on blood pressure in stroke-prone spontaneously hypertensive rats.

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The potential role of adrenaline, both circulating and in the central nervous system, in the maintenance of high blood pressure was examined in stroke-prone spontaneously hypertensive rats (SHRSP). alpha-Monofluoromethyldopa, a long-lasting inhibitor of dopa decarboxylase, was used to induce rapid

[Gastric lesions associated with acute stage of stroke. 2. Gastroendoscopic findings, urinary noradrenaline, adrenaline levels rate, plasma gastrin concentration and urinary excretion rate of 17-OHCS].

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Thyroid hemorrhage causing airway obstruction after intravenous thrombolysis for acute ischemic stroke.

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BACKGROUND There are several life-threatening complications associated with intravenous thrombolysis after acute ischemic stroke such as symptomatic intracerebral hemorrhage, orolingual angioedema, or less frequent, bleedings of the mucosa or ecchymosis. Aside from these known critical incidents,

Model-based prediction of the patient-specific response to adrenaline.

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A model for the cardiovascular and circulatory systems has previously been validated in simulated cardiac and circulatory disease states. It has also been shown to accurately capture the main hemodynamic trends in porcine models of pulmonary embolism and PEEP (positive end-expiratory pressure)

[Anaphylaxis needing adrenaline administration during anesthesia: a 7-year single-institution study].

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BACKGROUND Adrenaline is the key treatment for acute anaphylaxis; however, it is difficult to use it appropriately in terms of dosage and timing. If used incorrectly, adrenaline can cause cardiac infarction, stroke, recurrence and other problems. METHODS We collected data of suspected anaphylaxis

Haemodynamic effects of atenolol, pindolol and propranolol during adrenaline infusion in man.

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In a double blind, cross over study the haemodynamic effects of an i.v. infusion of adrenaline during concomitant administration of atenolol, pindolol, propranolol or placebo were examined in 7 healthy volunteers. During coadministration with placebo, adrenaline caused an increase in systolic blood

Adrenaline synthesizing nerve cells in the medulla of normotensive and hypertensive rats.

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1. We have studied the number and distribution of adrenaline synthesizing nerve cells in the medulla oblongata of the rat, using a combination of immunofluorescence to visualize the enzyme phenylethanolamine-N-methyltransferase (PNMT) and catecholamine fluorescence to detect central catecholamines.

Comparison of haemodynamic effects of intravenous isoprenaline and adrenaline after aortic valvar homograft replacement.

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The cardiovascular effects of an intravenous infusion of adrenaline (10 mug./min.) and isoprenaline (0.75 mug./min.) have been compared in two groups of six patients in the early post-operative period following aortic valvar homograft replacement. These doses were chosen to produce obvious

Cardiovascular effects of epidural local anaesthetics. Comparison of 0.75% bupivacaine and 0.75% ropivacaine, both with adrenaline.

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The cardiovascular effects of 20 ml 0.75% bupivacaine with adrenaline 5 micrograms/ml injected epidurally were compared with those of 20 ml 0.75% ropivacaine with adrenaline. Cardiovascular measurements were performed with a transthoracic electrical bioimpedance monitor. The maximum mean arterial

The role of the pericardium and the effects of adrenaline and changes in oxygen tension on the performance of an in situ perfused crocodile heart

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An in situ perfused crocodile (Crocodylus porosus) heart preparation was used to examine the mechanical responses of the heart to increases in adrenaline concentration, to a decrease in oxygen tension and to opening of the pericardium. Starling and power curves were constructed before and after

Effects of adrenaline on circulatory dynamics and cardiac function in rats administered chlorpromazine.

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We aimed to elucidate changes in circulatory dynamics and cardiac function during concomitant use of chlorpromazine (CPZ) and adrenaline (AD). An arterial line and left intraventricular pressure-volume measurement catheter were inserted in rats. CPZ 10 mg/kg was administered to the left great

Modification of the circulatory effects of extradural block combined with general anaesthesia by the addition of adrenaline to lignocaine solutions.

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The cardiovascular changes associated with extradural block were measured in two groups of patients who were also receiving light general anaesthesia. In the first two group (five patients) 2% plain lignocaine 20 ml was used and decreases occurred in mean arterial pressure, cardiac output, stroke

[The state of microcirculation and platelet hemostasis in patients with ischemic stroke after systemic thrombolytic therapy].

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OBJECTIVE The state of microcirculation and platelet hemostasis in patients with ischemic stroke after systemic thrombolytic therapy. METHODS Forty-two patients after systemic thrombolytic therapy (TLT) (mean age 64.26 [60.58; 68.06]) and 47 patients without TLT (54.52 [47.48; 60.83]) were studied.

Increased number of PNMT-immunofluorescent nerve cell bodies in the medulla oblongata of stroke-prone hypertensive rats.

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An antiserum to bovine adrenal PNMT was used to identify PNMT-containing nerve cell bodies in the medulla oblongata of 4-week-old normotensive Wistar-Kyoto rats and stroke-prone spontaneously hypertensive rats. The regional distribution of PNMT cells and the total number of PNMT cell profiles in

[Platelet hemostasis in patients with ischemic stroke after systemic thrombolytic therapy].

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OBJECTIVE To study the platelet aggregation (PA) in patients with ischemic stroke (IS) after systemic thrombolytic therapy (TLT). METHODS Twenty-four patients, including 13 with atherothrombotic IS and 11 with cardioembolic IS, have been examined after systemic TLT. Spontaneous and

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