arthus reaction/arginine

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Nitric oxide inhibits neutrophil infiltration in the reverse passive arthus reaction in rat skin.

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The role of nitric oxide (NO) in the reverse passive Arthus reaction elicited in the rat skin has been studied. The reverse passive Arthus reaction was modulated by test compounds given by intradermal injection in combination with anti-bovine serum albumin antibody. L-arginine (1.5-15 micromol/site)

Effect of nicotine-induced corticosterone elevation on nitric oxide production in the passive skin arthus reaction in rats.

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To elucidate the anti-inflammatory action of nicotine-induced corticosterone elevation on the passive skin Arthus reaction (PSAR), we investigated the inflammatory process in the PSAR. The polymorphonuclear leukocyte (PMNs) infiltration was observed just before as well as after elicitation by

Studies on the mechanisms involved in the inflammatory response in a reversed passive Arthus reaction in guinea-pig skin: contribution of neutrophils and endogenous mediators.

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1. Mediators of inflammation can increase vascular permeability in at least two different ways: by acting directly on endothelial cells or, indirectly, through an incompletely understood mechanism, dependent on circulating neutrophils. Neutrophil-dependent oedema formation has been described in the

Anti-inflammatory and vasoprotective activity of a retroviral-derived peptide, homologous to human endogenous retroviruses: endothelial cell effects.

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Malignant and inflammatory tissues sometimes express endogenous retroviruses or their proteins. A highly-conserved sequence from retroviral transmembrane (TM) proteins, termed the "immunosuppressive domain (ID)", is associated with inhibition of immune and inflammatory functions. An octadecapeptide

Cationic polyelectrolytes: potent opsonic agents which activate the respiratory burst in leukocytes.

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Bacteria and yeasts which are "opsonized" with cationic polyelectrolytes (poly-L-arginine, poly-L-histidine and arginine-rich histone) are avidly endocytosed by both "professional" and "non-professional" phagocytes. The cationized particles also strongly activate the respiratory burst in neutrophils

Absence of mast cell involvement in leukocyte adhesion and emigration induced by inhibition of nitric oxide synthase.

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Leukocyte adhesion on venules and their emigration to extravascular connective tissue are induced by administration of a nitric oxide synthase (NOS) inhibitor NG-nitro-L-arginine methyl ester. In the present study, the involvement of mast cells in the process was examined in genetically mast

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