lichen planus/phosphatase

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[Electron microscopic and histochemical research on lysosomes and acid phosphatase of epithelial cells of the normal oral mucosa and in lichen ruber planus].

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Expression of MHC class II antigens (HLA DR, DP and DQ) by keratinocytes in oral lichen planus.

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The incidence and distribution of keratinocytes expressing the major histocompatability complex antigens HLA DR, DP and DQ and their relationship to the density of the inflammatory infiltrate was determined in lesions of oral lichen planus and normal mucosa using an immunoalkaline phosphatase

Levels of potential oral cancer salivary mRNA biomarkers in oral cancer patients in remission and oral lichen planus patients.

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OBJECTIVE To gather preliminary data concerning the feasibility of using seven salivary mRNAs-IL-8; IL-1β; dual specificity phosphatase 1 (DUSP1); H3 histone family 3A (H3F3A); ornithin decarboxylase antizyme 1 (OAZ1); S100 calcium-binding protein P (S100P); and spermidine/spermine

Association between hepatitis C infection in Thai patients with oral lichen planus: A case-control study.

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OBJECTIVE The aim of the present study was to investigate the prevalence of hepatitis C virus (HCV) infection and evaluate liver enzyme levels in patients from upper northern Thailand with oral lichen planus (OLP). METHODS A case-control study of 101 patients with OLP and 101 patients without OLP

Histochemical identification of T cells in oral lichen planus.

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Lysosomal activity was demonstrated in the majority of the lymphocytes forming the mononuclear cell infiltrate in oral lichen planus. Staining for acid phosphatase, N-acetyl-beta-D-glucosaminidase, nonspecific esterase, and beta-glucuronidase was present in these cells in tissue sections, indicating

Immunohistochemical studies on vitronectin in elastic tissue disorders, cutaneous amyloidosis, lichen ruber planus and porphyria.

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Vitronectin, identical with serum-spreading factor and S-protein of complement, is a glycoprotein present in both plasma and tissue. It stimulates cell adhesion and spreading and affects the complement and coagulation pathways. Vitronectin immunoreactivity was recently found in conjunction with

Reduction of epithelial dendritic cells in keratotic lesion of oral lichen planus.

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The number and distribution of epithelial dendritic cells (EDC) in oral mucosal lesions are variable depending on the degree of epithelial maturation/differentiation and disease activity. Localized absence of EDC might impair mucosal immunologic protection, allow microbial colonization and enhance

Langerhans cell distribution and keratinocyte expression of HLADR in oral lichen planus.

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Keratinocyte expression of the Class II major histocompatibility complex antigen HLADR, is seen in several inflammatory disorders of skin and mucosa, including lichen planus. The purpose of this study is to determine whether the distribution of Langerhans cells and their expression of CD4 in oral

Hepatitis C virus infection in patients with oral lichen planus.

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BACKGROUND Lichen planus (LP) is a chronic mucocutaneous disease of uncertain etiology. Recent reports suggest that LP is an extrahepatic manifestation of Hepatitis C infection. OBJECTIVE To determine the association of Hepatitis C virus (HCV) infection with oral LP and to study the tests of liver

T cell subsets and macrophages in lichen planus. In situ identification using monoclonal antibodies and histochemical techniques.

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Skin lesions from 6 patients with lichen planus were studied for the presence of T cells and T cell subsets using monoclonal antibodies and indirect immunoperoxidase technique. Small numbers of Leu-2a-reactive suppressor-cytotoxic cells were present early in the basal cell layer in 2 patients with

Enzyme changes in lichen planus.

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Untreated cases of lichen planus have been studied by histochemical techniques. The acid phosphatase reaction in the transitional zone has been quantitatively estimated and compared with the adjacent relatively normal epidermis. It was found that despite a thickened and accentuated granular layer as

Lichen planus and liver disease: how strong is the association?

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One hundred and thirteen patients with histologically confirmed oral lichen planus, from three stomatology clinics, were examined for evidence of liver disease. No patient had clinical evidence of liver disease. Nine patients (7.9%) had a raised serum concentration of a single enzyme; 6 patients had

Epidemiological relationship of oral lichen planus to hepatitis C virus in an Indian population.

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OBJECTIVE The relationship between the hepatitis C virus (HCV) and oral lichen planus (OLP) remains a controversial subject for clinicians. Many studies aimed at studying the association between HCV and OLP have been conducted over the years. Geographical variations have been shown to be a major

The activation of Langerhans cells in oral lichen planus.

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The numbers of CD1, HLADR, HLADP and HLADQ positive, intraepithelial, dendritic cells were compared in lesions of oral lichen planus and normal oral mucosa using an immunoalkaline phosphatase technique. In normal mucosa, there were equal numbers of CD1 and HLADR positive cells but significantly

Histoenzymological features of epithelial cells in lesions of oral mucosa in cysts and ameloblastomas of jaws.

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A histoenzymological study was carried out on 41 tissue specimens removed at biopsy and for surgical operations of the following lesions: benign hyperkeratosis, lichen planus, severe epithelial dysplasia, carcinoma in situ, epidermoid carcinoma, radicular cyst, odontogenic keratocyst and

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