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meningoencephalitis/oedeem

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[The characteristics of the clinical manifestations of brain edema and swelling in bacterial meningoencephalitis].

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Based on the examination of 374 patients with bacterial meningoencephalitis, clinical forms (focal, general cerebral) and variants (progressing, pseudotumorous, protracted) of edema and swelling of the brain have been established. Incidence rate of their development was found to be dependent on
A biochemical study of the level of sialic acids in the blood of 60 patients and pathomorphological examination of autopsy material in 32 cases of meningoencephalitis of different etiology allowed to establish a certain relationship between the state of noncellular substance of vessel walls and the

[Generalized brain edema in non-purulent meningoencephalitis. The anti-edema effect of therapy with dexamethasone].

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We report about two female inpatients aged 58 and 24, suffering from non-purulent meningoencephalitis, in the first case caused by varizella-zoster-virus, in the second case probably due to viral infection. Both patients developed a diffuse brain edema associated with a progressive loss of
The authors investigated the quantitative content of a group of metals in the cerebrospinal fluid in 30 patients with bacterial meningoencephalitis with brain edema. It was established that selective permeability of the blood brain barrier has a compensatory character directed to maintenance of the
Nuclear absorption spectrometry was used to quantify concentrations of some metals in the liquor from 70 patients with bacterial meningoencephalitis and in different portions of the brain in 10 deceased patients. By calculation of the difference between dry and wet tissue mass after its drying the
Based on the evaluation done in 374 patients with bacterial meningoencephalitis there have been singled out clinical variants of spasmodic syndrome (generalized, incomplete, atypical, myoclonic), with their value being outlined in identifying brain edema and swelling as well as assessing the gravity

Neurogenic pulmonary edema following Cryptococcal meningoencephalitis associated with HIV infection.

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Neurogenic pulmonary edema (NPE) is a clinical syndrome characterized by the acute onset of pulmonary edema following a significant central nervous system insult. Only a few cases of NPE after Cryptococcal meningitis have been reported. We report a case of NPE following Cryptococcal

Optic Disc Edema in Glial Fibrillary Acidic Protein Autoantibody-Positive Meningoencephalitis.

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BACKGROUND Glial fibrillary acidic protein (GFAP) autoantibody-positive meningoencephalitis is a newly described entity characterized by a corticosteroid-responsive meningoencephalomyelitis. Some patients with GFAP autoantibody-positive meningoencephalitis have been found to have optic disc edema,

[Pathogenesis of cerebral edema-swelling in bacterial meningoencephalitis].

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Nuclear-absorption spectrometry was used to measure concentration of some metals in cerebrospinal liquid (CSL) of 70 patients with bacterial meningoencephalitis and in different brain compartments of 10 dead patients. Estimation of the difference between dry and wet tissue mass provided the picture

[Enzymologic characteristics of the course of brain edema in patients with meningococcal meningoencephalitis].

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[Clinico-morphological analysis of the neurological manifestations of brain edema in bacterial meningoencephalitis].

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Based on clinical and immunological examinations of 60 patients with MME and 30 normal persons, the dynamics of the blood CIC content was studied depending on the time and gravity of the disease. The discovered changes in argyrophilic membranes of the vascular walls are determined to a considerable

Fulminant Cytotoxic Edema in a Patient with Pneumococcal Meningoencephalitis.

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[A case of papillary edema during coxsackie B2 meningoencephalitis].

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[Acute Trypanosoma brucei gambiense meningoencephalitis detected by papillary edema].

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A 26 year old woman, living in Brazzaville, was referred by her ophthalmologist about papillo-oedema. Neurological examination showed frontal syndrome and papillo-oedema without another sign of intracranial hypertension. Electroencephalogram revealed bilateral delta waves and bifrontal points.
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