protein-energy malnutrition/tyrosine

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Serum mucoprotein (tyrosine) in protein calorie malnutrition.

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Altered macrophage intracellular signaling induced by protein-calorie malnutrition.

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Protein-calorie malnutrition (PCM) contributes to increased morbidity and mortality through impairment of host defense mechanisms and reduced macrophage function. The present study examined alterations in macrophage intracellular signaling associated with the impairment in host defense capabilities.

Tyrosyluria in marasmus.

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1. Plasma tyrosine and urinary p-hydroxyphenyl lactic acid (PHPLA) and p-hydroxyphenyl acetic acid (PHPAA) were studied in thirty patients with marasmus and twenty normal controls in the same age group. 2. In the control group conventional tyrosyluria was not observed but 30% of the group excreted

Biochemical study of malnutrition. Part V. Metabolism of phenylalanine and tyrosine.

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In order to elucidate the metabolic disorders which were observed for phenylalanine and tyrosine in protein-energy malnutrition, loads of both these amino acids were given to young mothers who showed all the clinical and biochemical symptoms of malnutrition and to healthy controls of the same age.

[Contribution of amino acid deficiency to primary hypothyroidism associated with protein-calorie malnutrition].

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A 68-year-old male patient without any previous thyroidal disease developed three times of transient primary hypothyroidism associated with protein-calorie malnutrition (PCM). Because of his diabetes mellitus, alcoholic hepatitis, chronic pancreatitis and blind loop syndrome, his nutritional balance

Plasma concentration of taurine is higher in malnourished than control children: differences between kwashiorkor and marasmus.

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Plasma free amino acids were determined in the plasma of severely malnourished children under two years of age. A total of thirty-one patients and eleven controls were evaluated: seventeen cases of kwashiorkor, eight cases of marasmus, and six cases of marasmic-kwashiorkor. Fasting plasma samples

Evaluation of protein-energy malnutrition in surgical patients from plasma valine and other amino acids, proteins, and anthropometric measurements.

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Eight plasma proteins, four anthropometric measurements, and 21 amino acids were measured in 24 fasting patients before surgery. A matrix of partial correlation coefficients, correcting for age and height, showed many correlations including several between biochemical and anthropometric data such as

Is the adult protein-energy malnutrition syndrome the same as that described in the infant?

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Protein-energy malnutrition, a multi-factorial disease, has been described predominantly in the infant. It was the aim of this research to give a biochemical assessment of the adult form and to compare it to the infantile syndrome within the same socio-cultural context of central Zaïre (Kwilu

Factors Associated With Protein-energy Malnutrition in Chronic Liver Disease: Analysis Using Indirect Calorimetry.

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We aimed to elucidate the incidence of protein-energy malnutrition (PEM) in patients with chronic liver disease and to identify factors linked to the presence of PEM.A total of 432 patients with chronic liver disease were analyzed in the current analysis. We defined patients with serum albumin level

Primary hypothyroidism in an adult patient with protein-calorie malnutrition: a study of its mechanism and the effect of amino acid deficiency.

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A man with diabetes mellitus, chronic hepatitis, chronic pancreatitis, and blind loop syndrome but without any previous thyroid disease developed three episodes of transient primary hypothyroidism associated with protein-calorie malnutrition (PCM). Clinical examinations suggested that this primary

Subclinical protein-energy malnutrition in under-privileged Ethiopian mothers and their newborn infants.

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An increased glycine/valine ratio in the maternal and cord vein plasma was observed in under-privileged women and their newborn babies in Ethiopia. There was a delay in the decrease of branch-chained amino acids during the immediate neonatal period. These findings indicate malnutrition in utero and

Branched-chain amino acid supplements reduced ascites and increased the quality of life in a patient with liver cirrhosis: A case report.

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Liver cirrhosis is frequently accompanied by malnutrition and hypoalbuminemia, which in turn commonly induces ascites in patients with liver cirrhosis. Ascites leads to abdominal distention and appetite loss, resulting in a deteriorated quality of life (QOL). Administration of branched-chain amino

Branched-chain amino acid-enriched nutrient increases blood platelet count in patients after endoscopic injection sclerotherapy.

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OBJECTIVE Protein and energy malnutrition is a severe problem for patients with liver cirrhosis (LC) and fasting often induces starvation which is a vitally important outcome. Dietary restriction is essential for endoscopic injection sclerotherapy (EIS) in patients with risky esophageal varices,

Effects of late evening snack including branched-chain amino acid on the function of hepatic parenchymal cells in patients with liver cirrhosis.

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OBJECTIVE A late evening snack (LES) improves protein-energy malnutrition due to overnight starvation and the catabolic state in patients with liver cirrhosis. Our aim was to examine whether LES including a branched-chain amino acid (BCAA) could maintain hepatic reserve and the function of hepatic

Experimental protein and energy deficiencies: effects on brain free amino acid composition in rats.

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1. The effects of protein-energy malnutrition on brain free amino acids of acidic and neutral groups were investigated in experimental rats. 2. Severe energy restriction did not modify the free amino acid composition of the brain while protein deficiency affected certain amino acids of the brain in

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