[The influence of dust and heating microclimate on lipid peroxidation].

In experimental pneumoconiosis both under normal and heating ambient temperature lipid peroxidation is not activated, although heating microclimate increases alteration of lung tissue and induces rapid and intensive pulmonary fibrosis. Association of dust and heating microclimate prolongs a period of "stress" myocardial affliction due to intensified lipid peroxidation and exhausted endogenous glutathione peroxidase, afterwards lipid peroxidation decreases with progressing sclerosis and fat degeneration in heart.