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Respiratory alkalosis in children with febrile seizures.
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OBJECTIVE
Febrile seizures (FS) are the most common type of convulsive events in children. FS are suggested to result from a combination of genetic and environmental factors. However, the pathophysiologic mechanisms underlying FS remain unclear. Using an animal model of experimental FS, it was
Experimental febrile seizures are precipitated by a hyperthermia-induced respiratory alkalosis.
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Febrile seizures are frequent during early childhood, and prolonged (complex) febrile seizures are associated with an increased susceptibility to temporal lobe epilepsy. The pathophysiological consequences of febrile seizures have been extensively studied in rat pups exposed to hyperthermia. The
Metabolic alkalosis, acute renal failure and epileptic seizures as unusual manifestations of an upside-down stomach.
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Upside-down stomach represents a critical and rare manifestation of hiatal hernias. Here we report on a 60-year-old male patient who was admitted to our hospital with epileptic seizures and dehydration. Laboratory tests revealed severe metabolic alkalosis (pH 7.56) with low potassium (2.7 mmol/l),
Alkalosis and seizure due to a cation-exchange resin and magnesium hydroxide.
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A patient with chronic renal failure received sodium polystyrene sulfonate and magnesium hydroxide for the treatment of hyperkalemia. This combination therapy produced a severe metabolic alkalosis which, in the presence of the patient's chronic hypocalcemia, precipitated a grand mal seizure.
A 3-year old girl with seizures, hypokalemia and metabolic alkalosis.
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A 3-year-old girl presented to the emergency department with seizures, low-grade fever and vomiting. She had tachycardia and a slow capillary refill. Blood pressure could not be measured. Because of suspected sepsis and/or meningo-encephalitis, broad spectrum antibiotics and antiviral medication
Brain alkalosis causes birth asphyxia seizures, suggesting therapeutic strategy.
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OBJECTIVE
The mechanisms whereby birth asphyxia leads to generation of seizures remain unidentified. To study the possible role of brain pH changes, we used a rodent model that mimics the alterations in systemic CO(2) and O(2) levels during and after intrapartum birth asphyxia.
METHODS
Neonatal rat
Acid extrusion via blood-brain barrier causes brain alkalosis and seizures after neonatal asphyxia.
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Birth asphyxia is often associated with a high seizure burden that is predictive of poor neurodevelopmental outcome. The mechanisms underlying birth asphyxia seizures are unknown. Using an animal model of birth asphyxia based on 6-day-old rat pups, we have recently shown that the seizure burden is
Study of blood-brain tissue [H+] gradient during respiratory alkalosis and experimental seizures in newborn piglets. Effect on phenobarbital distribution.
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Hypochloremic alkalosis with symptoms of seizure disorder.
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Respiratory alkalosis: "basic" mechanism of febrile seizures?
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Shock, seizures, and coma with alkalosis during mechanical ventilation.
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Pica presenting as metabolic alkalosis and seizure in a dialysis patient.
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Severe metabolic alkalosis due to baking soda ingestion: case reports of two patients with unsuspected antacid overdose.
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Oral ingestion of baking soda (sodium bicarbonate) has been used for decades as a home remedy for acid indigestion. Excessive bicarbonate ingestion places patients at risk for a variety of metabolic derangements including metabolic alkalosis, hypokalemia, hypernatremia, and even hypoxia. The
Infantile hypertrophic pyloric stenosis presenting as pseudo-Bartter's syndrome and seizures: report of one case.
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We report a hypertrophic pyloric stenosis case with an unusual initial presentation of seizures and Bartter's syndrome like symptoms. This case suffered from vomiting, diarrhea and poor appetite for several days, and seizures developed after these symptoms. From laboratory tests, hypochloremic and
Two adjacent mutations on chromosome 16 discovered in a patient presenting with generalized convulsions after influenza A virus infection.
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A 49-year-old otherwise healthy man was admitted to our hospital because of repeated generalized convulsions after influenza A virus infection. His family history was notable for consanguinity of parents. Initial laboratory tests revealed metabolic alkalosis with hypomagnesemia, as well as an
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