allantoin/atrophy

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The effect of allantoin on cellular multiplication in degenerating and regenerating nerves.

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Injury to a peripheral nerve is, among other things, followed by degeneration of axons and myelin, as well as by a sharp increase in the number of cells (especially Schwann cells) in the part distal to the injury. The effect of allantoin--a cell proliferant--was tested on the above-mentioned

Effects of a rhizome extract of Dioscorea batatas and its bioactive compound, allantoin in high fat diet and streptozotocin-induced diabetic mice and the regulation of liver, pancreas and skeletal muscle dysfunction.

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Dysfunction of glucose metabolism is associated with the occurrence of metabolic syndromes, including type 2 diabetes mellitus (T2DM). In this study, we investigated the anti-diabetic effects of yam extract and allantoin in high-fat-diet (HFD) and streptozotocin (STZ)-induced diabetic

Metabolomics Coupled with Transcriptomics Approach Deciphering Age Relevance in Sepsis.

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Sepsis is a severe disease frequently occurred in the Intenisive Care Unit (ICU), which has a very high morbidity and mortality, especially in patients aged over 65 years. Owing to the aging effect and the ensuing deterioration of body function, the elder patients may have atypical responses to

Renal Calculi (Uric Acid, Hyperuricosuria, Gouty Diathesis)

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Uric acid, a heterocyclic compound, is produced as a byproduct of purine metabolism. Uric acid can be generated from endogenous as well as exogenous sources. The human body makes about 300-400 mg of uric acid per day, with most of it being built in the liver. Purines degenerate into inosine and

[Nitrogen metabolism and its control mechanisms].

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N intake in the form of protein has neither got an upper nor a lower limit for agricultural working animals within a diet and there is no control mechanism for it. A high surplus of certain amino acids results in a reduction of feed intake. N excretion in faeces depends on 1) the excretion of N

1H NMR-based metabolomics study on repeat dose toxicity of fine particulate matter in rats after intratracheal instillation.

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Systemic metabolic effects and toxicity mechanisms of ambient fine particulate matter (PM2.5) remain uncertain. In order to investigate the mechanisms in PM2.5 toxicity, we explored the endogenous metabolic changes and possible influenced metabolic pathways in rats after intratracheal instillation

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