muscular atrophy/neoplasms

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Biphasic intra-abdominal desmoplastic small cell tumor in a patient with proximal spinal muscular atrophy.

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A case is reported of intra-abdominal desmoplastic small cell tumor (IDSCT) with biphasic histologic features in a patient with proximal spinal muscular atrophy. The tumor was composed of small epithelial cell nests with spindle cell sarcomatous areas. Both areas were surrounded by a desmoplastic

A case of spinal and bulbar muscular atrophy with high-stage and high-Gleason Score prostate cancer responded to maximal androgen blockade therapy.

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Spinal and bulbar muscular atrophy is a hereditary motor neuron disease caused by the expansion of CAG triplets in the androgen receptor. Because the length of CAG repeat is inversely related with androgen receptor function, patients with a longer CAG repeat are expected to have a lower incidence of

Skeletal muscular atrophy in malignant tumour diseases: neurogenic or unspecific?

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Muscles from patients who had died from malignant tumours were examined with regard to changes in the intramuscular nerve twigs and the axon terminals or the motor end-plates, respectively. Three muscles from each of 120 cases were obtained at autopsy and stained with conventional methods. Nineteen

[Tumor-like transformation of the median nerve in neural muscular atrophy].

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This paper reports a case of local enlargement of the median nerve in the upper arm due to the very rare condition of hypertrophic neuropathy. A characteristic feature of this affection of peripheral nerves, the cause of which is unknown, is the onion-skin-like hypertrophy of the Schwann cells and

[Case of spinal cord neoplasm with raised intracranial pressure and upside-down champagne bottle shaped muscular atrophy].

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BRCA2-Associated Prostate Cancer in a Patient With Spinal and Bulbar Muscular Atrophy.

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[Diagnostic significance of central muscular atrophy in tumors and vascular diseases of the brain].

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CAG repeats in the androgen receptor: a case of spinal and bulbar muscular atrophy associated with prostate cancer.

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Exonic trinucleotide repeats and expression of androgen receptor gene in spinal cord from X-linked spinal and bulbar muscular atrophy.

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We studied exonic trinucleotide repeats and expression of androgen receptor (AR) gene in the spinal cord from an autopsied patient with X-linked spinal and bulbar muscular atrophy (SBMA). Forty-nine CAG triplet repeats were found in tissues from the spinal cord, cerebrum, cerebellum, cardiac muscle

S137 phosphorylation of profilin 1 is an important signaling event in breast cancer progression.

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BACKGROUND Profilins are actin-modulating proteins regulating many intracellular functions based on their multiple and diverse ligand interactions. They have been implicated to play a role in many pathological conditions such as allergies, cardiovascular diseases, muscular atrophy, diabetes,

Exercise as a prescription therapy for breast and colon cancer survivors.

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BACKGROUND Breast and colon tumors are the most common types of cancer in the general population. As a result of improved diagnosis and treatment, more people are now surviving cancer. Lifestyle has been identified as one of the potential risk factors for cancer, and it has been demonstrated

[Energy metabolism in cancer patients].

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The presence of cachexia as defined by a series of clinical symptoms, such as anorexia, weight loss, muscular atrophy, tissue wasting, altered organ function, is frequently observed in cancer and makes a decisive contribution to morbidity and mortality. The onset of neoplastic cachexia is

Peripheral neurofibromatosis and peroneal muscular atrophy.

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We studied four patients with peripheral neurofibromatosis and a neuropathy that had the clinical characteristics of peroneal muscular atrophy. Nerve conduction velocities were slowed by less than 40% of normal, and electromyography demonstrated denervation. Sural nerve biopsies from two patients,

Intramedullary astrocytoma presenting as spinal muscular atrophy.

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We present a 6-year-old patient with a spinal cord tumor who had been followed with the diagnosis of spinal muscular atrophy since the age of 23 months. Reasons for reevaluating the diagnosis of spinal muscular atrophy were the early onset of scoliosis, the slight asymmetry in weakness of

[An extramedullary spinal cord tumor associated with syringomyelia: a case report].

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A 53-year-old woman presenting weakness in the left hand for ten years was diagnosed seven years ago as having juvenile muscular atrophy of the unilateral upper extremity. Afterwards, her left forearm and hand gradually became weaker. A neurologist pointed out syringomyelia in the film of MR

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