brain edema/nicotina

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[Effect of several neuroleptic, adreno-, sympatho- and cholinolytic substances on the development of experimental cerebral edema induced by nicotine].

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The influence of some neuroleptic, adreno, sympatho- and cholinolytic substances on the development of experimental brain edema induced with nicotine was studied in tests conducted on rats. It was ascertained that marked antiedemic properties display drugs blocking the alpha-adrenoreceptors

Nicotine exacerbates brain edema during in vitro and in vivo focal ischemic conditions.

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We have previously shown that nicotine, the addictive component of tobacco products, alters the blood-brain barrier (BBB) Na(+),K(+),2Cl(-) cotransporter (NKCC) during in vitro hypoxia-aglycemia exposure. Attenuation of abluminal NKCC suggests that accumulation of ions in the brain extracellular

Chronic nicotine pretreatment protects the blood-brain barrier against nicotine-induced seizures in the rat.

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This study was designed to investigate the possible protective actions of nicotine on cerebrovascular permeability in convulsions during nicotine-induced seizures. We have measured the permeability changes in the blood-brain barrier (BBB) macroscopically and spectrophotometrically by using Evans

Transdermal nicotine replacement therapy in cigarette smokers with acute subarachnoid hemorrhage.

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BACKGROUND We evaluated the safety of nicotine replacement therapy (NRT) in active smokers with acute (aneurysmal) subarachnoid hemorrhage (SAH). METHODS A retrospective observational cohort study was conducted in a prospectively collected database including all SAH patients admitted to an 18-bed

[Intensity of glycolysis with brain edema in rats and application of some pharmacological agents].

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In the in vitro experiments the state of glycolytic processes was studied in the rat brain tissues with development of edema induced by injury and administered nicotine and monoiodoacetate as well as with preliminary introduction of phentolamine, benactyzine, aminazine and trifluoperazine. The brain

Therapeutic effect of nicotine in a mouse model of intracerebral hemorrhage.

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Intracerebral hemorrhage (ICH) resulting from the leakage of blood into the brain parenchyma triggers severe tissue damage involving neurodegeneration and inflammation. Increasing lines of evidence indicate that the stimulation of central nicotinic acetylcholine receptors affords neuroprotection

Mito-Tempo prevents nicotine-induced exacerbation of ischemic brain damage.

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Nicotine may contribute to the pathogenesis of cerebrovascular disease via the generation of reactive oxygen species (ROS). Overproduction of ROS leads to brain damage by intensifying postischemic inflammation. Our goal was to determine the effect of Mito-Tempo, a mitochondria-targeted antioxidant,

A controlled study of the relationship between Bordetella pertussis infections and sudden unexpected deaths among German infants.

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OBJECTIVE This was a prospective, controlled, multicenter study to investigate the relationship between Bordetella pertussis infections and sudden unexpected deaths among German infants. METHODS Between 1995 and 1997, all infants who died at 7 to 365 days of age and for whom autopsies were performed

Acute Pancreatitis and Posterior Reversible Encephalopathy Syndrome: A Case Report.

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The posterior reversible encephalopathy syndrome is a neurological syndrome characterized by headache, confusion, visual disturbances and seizures associated with identifiable areas of cerebral edema on imaging studies. The authors report the case of a man, 33 years-old, leukodermic with a history

Stroke is an emergency.

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Stroke is an emergency. Ischemic stroke is similar to myocardial infarction in that the pathogenesis is loss of blood supply to the tissue, which can result in irreversible damage if blood flow is not restored quickly. Public education is needed to emphasize the warning signs of stroke. Patients

α7 Nicotinic acetylcholine receptor agonist attenuates neuropathological changes associated with intracerebral hemorrhage in mice.

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We have demonstrated previously that nicotine affords neuroprotective and anti-inflammatory effects against intracerebral hemorrhage (ICH)-associated neuropathological changes. The present study was undertaken to clarify whether subtype-specific agonists of nicotinic acetylcholine receptors (nAChRs)

Cranioplasty after decompressive hemicraniectomy: underestimated surgery-associated complications?

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OBJECTIVE Decompressive hemicraniectomy (DC) and duroplasty after malignant brain infarction or traumatic brain injury is a common surgical procedure. Usually, preserved bone flaps are being reimplanted after resolution of brain swelling. Alloplast cranioplasties are seldom directly implanted due to

Drugs of abuse and blood-brain barrier endothelial dysfunction: A focus on the role of oxidative stress.

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Psychostimulants and nicotine are the most widely abused drugs with a detrimental impact on public health globally. While the long-term neurobehavioral deficits and synaptic perturbations are well documented with chronic use of methamphetamine, cocaine, and nicotine, emerging human and experimental

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