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chlamydia infections/triacylglycerol
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Cardiac heart disease in the era of sucrose polyester, Helicobacter pylori and Chlamydia pneumoniae.
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Recent evidence associates inflammatory mediators with coronary heart disease. Elevation of acute-phase reaction (APR) proteins such as serum amyloid A, fibrinogen, CRP and haptoglobin in response to Helicobacter pylori (H. pylori) infection was shown to initiate gastritis and ischemic heart
Markers of Chlamydia pneumoniae and human cytomegalovirus infection in patients with chronic peripheral vascular disease and their relation to inflammation, endothelial dysfunction and changes in lipid metabolism.
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Our aim was to detect markers of Chlamydia pneumoniae (CPN) and human cytomegalovirus (HCMV) infection in patients with peripheral vascular occlusive disease and to follow markers of inflammation, endothelial dysfunction and lipid metabolism alteration in patients with active infection. CPN genome
[Occurrence of chlamydia infection in relation to lipidemia indicators in the etiology of unstable angina pectoris].
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The presence of Chlamydia pneumoniae infection was examined in 66 patients with unstable angina pectoris (UAP), 155 patients with acute myocardial infarction (AIM) and 112 controls without signs of a heart disease. Besides evaluation of anamnestic data, ECG and coronarographic examination, serologic
[Chlamydia pneumoniae and the risk of ischaemic heart disease. Prevalence in a group of hospitalized elderly men].
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Chlamydia pneumoniae (CP) causes frequently nosocomial pneumonia and other inflammations of the upper and lower airways. Initially reports on the association between infection with CP and ischaemic heart disease (IHD) were published in 1988 by Saikku et al. who found a higher antibody titre against
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