adenyl cyclase/hypoxia

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[Effect of adaptation to high-altitude hypoxia on the adrenoreactivity of the heart and the state of the adenyl cyclase and phosphodiesterase systems of the myocardium].

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Experiments in white rats suggested that changes in activity of certain intracellular systems played a significant role in the mechanism of increasing adrenoreactivity of the heart in adaptation to high-altitude hypoxia.

[Morphological study of the adenyl cyclase activity of the rat heart in experimental hypoxia and ischemia].

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Alteration of the activity and localization of the reaction of adenylcyclase after perfusion of the rat heart with nitrogen, carbon monoxide and after ischaemia was studied by electronmicroscopy. In the first minutes of hypoxia increase of the activity of the adenylcyclase was revealed. After

[Activity of adenyl cyclase in rat brain under conditions of hypoxia].

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[Effect of adaptation to high altitude hypoxia and cold on rat myocardial adenyl cyclase activity].

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Effect of hypoxia on the cerebral energy state.

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The effect of hypoxia on glycogen content, adenyl cyclase activity, cyclic 3',5'-AMP concentration, lactate: pyruvate ratio and the energy charge potential connected with the pool of adenylates was evaluated in the motor area of the cortex of beagle dogs. At first, controlled hypoxia induces

Adenyl cyclase system and cerebral energy state.

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The energy charge potential of the cerebral adenylate system, the cerebral lactate: pyruvate system, and the cerebral glycogen level were used to characterize the cerebral energy state of the brain. Cerebral adenyl cyclase activity and cyclic AMP concentrations were investigated to evaluate their

Interactions of adenosine, prostaglandins and nitric oxide in hypoxia-induced vasodilatation: in vivo and in vitro studies.

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Adenosine, prostaglandins (PG) and nitric oxide (NO) have all been implicated in hypoxia-evoked vasodilatation. We investigated whether their actions are interdependent. In anaesthetised rats, the PG synthesis inhibitors diclofenac or indomethacin reduced muscle vasodilatation evoked by systemic

Mediated (nonactive) transport of glucose in Mammalian cells and its regulation.

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Mediated (nonactive) transport of glucose in mammalian cells is characterized by saturation kinetics, stereospecificity, sensitivity to inhibition by phlorizin and certain sulfhydryl-blocking agents, a temperature coefficient of about 2, an inability to utilize metabolic energy, and

Insulin: fundamental mechanism of action and the heart.

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Insulin accelerates the entry of glucose and amino acids into muscle cells by acting upon the 'carrier-facilitated' transport mechanism. For glucose this process is passive and leads to equilibration of intracellular and extracellular concentrations. In heart muscle, glucose transport is a

Experimental Observations on the Biological Significance of Hydrogen Sulfide in Carotid Body Chemoreception.

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The cascade of transduction of hypoxia and hypercapnia, the natural stimuli to chemoreceptor cells, is incompletely understood. A particular gap in that knowledge is the role played by second messengers, or in a most ample term, of modulators. A recently described modulator of chemoreceptor cell

Induction of division synchrony in Tetrahymena pyriformis by a single hypoxic shock. Its use in elucidating control of the cell cycle by adenosine 3':5'-monophosphate.

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A single hypoxic shock was used to induce division synchrony in Tetrahymena pyriformis. Hypoxia results in accumulation of the cells in the G2 phase of the cell cycle. Cyclic AMP and adenyl cyclase activity were measured in this system. Cell-cycle blockade was associated with extraordinarily high

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