dystonia/infarction

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[Dystonia caused by putamino-capsulo-caudate infarction in a child].

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Three cases of stroke in childhood are reported. In the 3 cases, an infarct involved the territory of the striato-lenticular arteries. Clinically, there was a pure motor hemiplegia and a dystonia which appeared while the hemiplegia was disappearing. The dystonic syndrome due to stroke appears only

Subthalamic Nucleus Deep Brain Stimulation in Post-Infarct Dystonia

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Dystonia secondary to cerebral infarcts presents months to years after the initial insult, is usually unilateral and causes significant morbidity. Deep brain stimulation (DBS) of the globus pallidus internus (GPi) is established as the most frequent target in the management of the dystonic symptoms.

Dystonia and tremor secondary to thalamic infarction successfully treated with thalamotomy of the ventralis intermedius nucleus.

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BACKGROUND Focal thalamic lesions have been associated with a variety of involuntary movements such as tremor, dystonia, and chorea-ballism. METHODS We describe a patient with severe hyperkinesias of the right arm secondary to a thalamic infarction in the left postero-ventral region of the

Sensorimotor hemiparesis with secondary cervical dystonia following lateral caudal medullary infarction without signs and symptoms of Wallenberg syndrome.

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We report the case of an 84-year-old woman who suddenly developed motor and both superficial and deep sensory hemiparesis on the left side, and cervical dystonia with a head tilt to the right side. A brain MRI showed an infarct in the left lateral caudal medulla. It is clinically important to

[Comparative study of the exercise tolerance of patients with autonomic circulatory dystonia, angina pectoris and previous myocardial infarction].

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Ninety-eight men were examined. Of these, 13 were practically healthy, 29 had vegetovascular dystonia, 15 stable angina pectoris, and 41 men suffered myocardial infarction. Each group was appraised for work fitness. The double product, work, chrono- and inotropic reserves of the heart, as well as

Basal ganglia infarction as a possible cause of cervical dystonia.

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Cervical dystonia (CD) is usually an idiopathic disorder that results in abnormal movements and painful postures of the neck. Although symptomatic CD caused by focal CNS lesions has been described in the literature, it is an exceedingly rare phenomenon. We report two women who had an abrupt onset of

[Virtual dystonia due to posterior ventrolateral thalamic infarct: case report].

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Behaviors, actions and movements may take place as purely mental events, as in the obsessions of obsessive-compulsive disorder, phantom limbs or sensory tics. In the present paper we report on the case of a 43-year-old diabetic hypertensive man who developed an incomplete form of the Dejerine-Roussy

[A case of post-hemiplegic painful dystonia following thalamic infarction with good response to botulinus toxin].

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We report a 67-year-old hypertensive right-handed woman who developed severe pain and dystonia in her left upper and lower extremity after a thalamic infarction. She was well until 9 months prior to the present admission to our hospital, when she had an acute onset of left hemiparesis which turned

Focal Leg Dystonia Associated with Cerebellar Infarction and Application of Low-Frequency Cerebellar Transcranial Magnetic Stimulation: Evidence of Topographically Specific Cerebellar Contribution to Dystonia Development.

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The cerebellum has recently been highlighted as a key neural substrate responsible for dystonia. A 57-year-old female presented with isolated focal leg dystonia that developed 8 years after acute cerebellar infarction. Brain magnetic resonance imaging showed an old cerebellar infarct in the right

Acute Onset Dystonia after Infarction of Premotor and Supplementary Motor Cortex.

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OBJECTIVE Poststroke dystonia is the second most common movement disorder after chorea and often has a delayed manifestation. Lesions of the contralateral lenticular nucleus, particularly the putamen, have been implicated in the pathogenesis of dystonia. We present an unusual case of rapid onset of

Early-onset dystonia after supplementary motor area infarction.

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A 63-year-old patient with the right supplementary motor area infarct developed early-onset dystonia in the left upper extremity. The mechanisms involved in the genesis of focal dystonia are discussed with emphasis on cortical basal ganglia circuit and efferent projections from the supplementary

Cervico-shoulder dystonia following lateral medullary infarction: a case report and review of the literature.

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BACKGROUND Secondary cervical dystonia is induced by organic brain lesions involving the basal ganglia, thalamus, cerebellum, and brain stem. It is extremely rare to see cervical dystonia induced by a medullary lesion. METHODS We report a case of an 86-year-old Japanese woman who developed cervical

Action hand dystonia after cortical parietal infarction.

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A 46-year-old patient with a pure left cortical infarct affecting mainly the gyrus postcentralis developed action dystonia in the right hand. Mechanisms involved in the genesis of focal secondary dystonia are discussed with emphasis on abnormal cortical sensory processing.

Focal dystonia in association with cerebral infarction.

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Blepharospasm and jaw closing dystonia after parietal infarcts.

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