epilepsia partialis continua/hemoragie

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Epilepsia partialis continua possibly caused by cerebellar lesion.

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The cerebellum is known to have inhibitory effects on seizures. Nevertheless, cerebellar dysplastic lesions can be epileptogenic. We report a patient presenting with epilepsia partialis continua (EPC) following a hemorrhagic cerebellar lesion. Possible pathophysiological mechanisms are discussed.

Alien hand syndrome after epilepsia partialis continua: FDG PET and MRI studies.

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Epilepsia partialis continua (EPC) is clinically defined as a syndrome of continuous focal jerking of a body part, usually a distal limb, occurring over hours, days, or even years. It is considered the status epilepticus equivalent of simple partial motor seizures. A 48-year-old right-handed man

[Psychopathological symptoms in atypical viral hemorrhagic tick-borne encephalitis].

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A 17 year old boy was admitted because of symptoms of a catatonic syndrome. During the diagnosis we ascertained that there was bleeding from the central nervous system of unknown origin. The intensification of neurological and general symptoms/among others-hyperthermia/suggested haemorrhagic

Acute stroke-elicited epilepsia partialis continua responsive to intravenous sodium valproate.

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We present an interesting case of epilepsia partialis continua (EPC) responsive to valproate treatment as an acute manifestation of cortical ischaemia in the course of vasospasm after subarachnoid bleeding A 72-year-old, right-handed woman suffering a subarachnoid haemorrhage complicated with

[Epilepsia partialis continua (Kojevnikov's syndrome)].

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BACKGROUND Lesions close to the central sulcus may give rise to focal motor seizures of long duration. This condition is called epilepsia partialis continua (Kojevnikov's syndrome). METHODS Over the last two years, the National Centre for Epilepsy in Norway has treated 12 patients with epilepsia

A unique case of nontraumatic femoral neck fracture following epilepsia partialis continua.

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People with epilepsy are more accident prone than the non-epilepsy population. Bone fractures are most often due to seizure-related falls. However, seizures themselves, in particular generalized tonic-clonic seizures, may also cause fractures, e.g. of the thoracic spine. Here, I present a man who

Focal neuronal loss, reversible subcortical focal T2 hypointensity in seizures with a nonketotic hyperglycemic hyperosmolar state.

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BACKGROUND Neuroimaging in seizures associated with nonketotic hyperglycemia (NKH) is considered normal. We report magnetic resonance imaging (MRI) abnormalities in four patients with NKH and seizures. METHODS We prospectively evaluated clinical and radiological abnormalities in four patients with

[Status epilepticus in children. Study of 70 cases].

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We presented the experience at the Emergency Unit of the National Institute of Pediatrics with children with Status Epilepticus (SE). This series studied 70 patients, the greatest frequency was seen among infants (55%), followed by preschool children (17%). The most frequent type of SE was

Immediate Transfer for Clot Extraction in a Young Woman With Leukemia and Asparaginase-Associated Acute Cerebral Vein Thrombosis.

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We present the case of an 18-year-old woman with B-cell acute lymphoblastic leukemia (ALL) who developed hemorrhagic stroke and epilepsia partialis continua due to acute cerebral vein thrombosis (CVT). The patient had 10 risk factors for CVT (including use of asparaginase chemotherapy for the ALL)

Dexamethasone as Abortive Treatment for Refractory Seizures or Status Epilepticus in the Inpatient Setting.

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Refractory seizures or status epilepticus (RS/SE) continues to be a challenge in the inpatient setting. Failure to abort a seizure with antiepileptic drugs (AEDs) may lead to intubation and treatment with general anesthesia exposing patients to complications, extending hospitalization, and

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