focal nodular hyperplasia/phosphatase

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Orthotopic liver transplantation in glucose-6-phosphatase deficiency--Von Gierke disease--with multiple hepatic adenomas and concomitant focal nodular hyperplasia.

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Von Gierke disease is occasionally complicated by hepatic adenomas (HAs) causing great concern owing to the current difficulties in monitoring them regarding malignant transformation. Orthotopic liver transplantation (OLT) is proposed as a therapeutic tool when multiple HAs and poor metabolic

Pregnancy complicated by focal nodular hyperplasia: a case report of one woman over two consecutive courses of pregnancy.

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UNASSIGNED We encountered a woman with a preexisting large focal nodular hyperplasia (FNH) of the liver, persisting during two separate pregnancies. FNH size was not affected by either pregnancy. Her elevated serum γ-glutamyltransferase and alkaline phosphatase levels before pregnancy were reduced

Type La glycogen storage disease with focal nodular hyperplasia in siblings.

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Glycogen storage disease type I (GSD-I) is an inherited disorder that is due to a glucose-6-phosphatase (G6Pase) deficiency. There have been recent reports of hepatocellular tumors in adults with this disease. Hepatic adenoma is the most common tumor described but others, including hepatocellular

Hepatocellular carcinoma and focal nodular hyperplasia of the liver in a glycogen storage disease patient.

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Glycogen storage disease type Ia (GSD-Ia; also called von Gierke disease) is an autosomal recessive disorder of carbohydrate metabolism caused by glucose-6-phosphatase deficiency. There have been many reports describing hepatic tumors in GSD patients; however, most of these reports were of

Alkaline phosphatase activity in human and rat liver tumors.

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Enzyme activity measurements of alkaline phosphatase in surgically removed human liver tumors showed elevated level of the enzyme in 6 focal nodular hyperplasias, reduction in 8 primary hepatocellular carcinomas, and no change in the 4 adenoma samples. The activity represented liver type of alkaline

Focal hepatic glycogenosis.

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Foci of altered hepatocytes (FAH) including clear cell foci excessively storing glycogen (focal hepatic glycogenosis) are well known as preneoplastic lesions in animal models of hepatocarcinogenesis induced by chemical, physical or viral agents. The occurrence of similar lesions has been studied in

[Hepatocellular adenoma after oral contraception].

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Raised serum basic phosphatase was found incidentally in a woman aged 43 years. Investigation with biopsy revealed a hepatocellular adenoma. The tumour regressed after withdrawal of Neogentrol oral contraception which the patient had consumed for 17 years. The patient did not desire invasive

Perioperative management of benign hepatic tumors in patients with glycogen storage disease type Ia.

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Glycogen storage disease type Ia (GSD-Ia; von Gierke disease) is an inherited disorder caused by glucose-6-phosphatase deficiency, and there have been some reports of hepatic tumors in patients with this disease. We report two patients with benign hepatic tumors with GSD-Ia. One is a 19-year-old man

Long-term hepatic outcomes in survivors of stage 4S and 4 neuroblastoma in infancy.

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BACKGROUND Infants with stage 4 and 4S neuroblastoma (NB) have a superior prognosis to older children. However, they often require intensive therapy including abdominal radiation. We aimed to investigate the long-term hepatic outcomes in infants with stage 4S and 4 NB. METHODS We reviewed the charts

[Nodular regenerative hyperplasia of the liver associated with a CREST syndrome with multiorgan involvement].

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A 52 year old woman with CREST syndrome (calcinosis, Raynaud's phenomenon, esophageal hypomotility and telangiectasia) and visceral involvement of the kidneys, lungs and liver is presented. The hepatic implication was a focal nodular hyperplasia of the liver which is a component not frequently

Clinical trials with a new iodinated lipid emulsion for computed tomography of the liver.

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A new iodinated lipid emulsion, Intraiodol, for which animal studies have indicated better tolerance than for other iodinated lipid emulsions, was tested in 15 patients with malignant lesions, and in one patient with focal nodular hyperplasia. Repeated CT scans of the liver and spleen and blood

[Focal hepatic peliosis without evident aetiology].

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Hepatic peliosis is a rare vascular disorder of the liver characterized by small cavities containing blood. It is most often asymptomatic but may give rise to portal hypertension and even fatal liver rupture. In this case a 47-year-old woman presented with abdominal pain and elevated alkaline

A triple stain of reticulin, glypican-3, and glutamine synthetase: a useful aid in the diagnosis of liver lesions.

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BACKGROUND The correct histologic diagnosis of mass lesions of the liver can be difficult, especially in biopsy samples. Reticulin, glypican-3, and glutamine synthetae are stains that can help distinguish hepatocellular carcinoma, hepatic adenoma, and focal nodular hyperplasia. OBJECTIVE To evaluate

[Spontaneous regression of a large liver tumor with markedly elevated serum enzyme levels].

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The case of a liver tumor of 10 cm diameter in a 32-year-old asymptomatic woman is described with markedly elevated liver enzymes in the serum (transaminases and alkaline phosphatase). The tumor corresponded to a focal nodular hyperplasia or a hepatic adenoma. The regression of this hepatic tumor

Marker Enzymes of Rat Chemical Hepatocarcinogenesis in Human Liver Tumors.

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Reduced glucose-6-phosphatase, increased GGT activity and reduction of cytochrome P-450 content are considered to be markers of chemical hepatocarcinogenesis in rats. The significance of these changes were studied in certain human liver lesions; adenoma, focal nodular hyperplasia and hepatocellular

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