hypophosphatemia/obezitate

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Prevalence of overweight and obesity in children with X-linked hypophosphatemia.

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X-linked hypophosphatemia (XLH) is a rare disease characterized by low phosphate levels. Scientific evidence points to link between hypophosphatemia and obesity in general population. The aim of our longitudinal observational study was to investigate the prevalence of obesity and

Effect of propranolol on hypophosphatemia in overweight.

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A negative correlation was found in 54 hypertensive overweight patients between the excess body weight and the level of serum inorganic phosphorus. The effect of propranolol upon the serum concentration of phosphorus, sodium, potassium, calcium and magnesium in 20 of the above mentioned patients as

Enhanced postglucose hypophosphatemia during starvation therapy of obesity.

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[Hypophosphatemia in the hospitalized patient].

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BACKGROUND Phosphate is an obligatory component of cellular function. Serum, levels are maintained within narrow levels by regulatory mechanisms. The normal range of phosphate concentration is 2.5-4.5 mg/dL. In certain clinical situations these mechanisms are insufficient and hypophosphatemia

Risk of cardiovascular involvement in pediatric patients with X-linked hypophosphatemia.

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To find out if cardiovascular alterations are present in pediatric patients with X-linked hypophosphatemia (XLH).Multicentre prospective clinical study on pediatric patients included in the RenalTube database ( www.renaltube.com ) with genetically confirmed

The effect of osteotomy on bowing and height in children with X-linked hypophosphatemia.

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The clinical records of 40 patients with X-linked hypophosphatemia who were treated and followed for at least 36 months were examined retrospectively. The patients were divided into those treated with medication only (group A) and those treated with medication and surgery (group B). At follow-up,

[Severe hypophosphatemia in intensive care].

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Phosphorus plays an essential role in cellular metabolism, especially in the oxidative phosphorylation process and in the synthesis of 2-3 DPG and membrane phospholipids. Moreover phosphorus is necessary as a buffer, mainly when the organism's principal buffer, the H2CO3/HCO3- system, is working at

Treatment of hypophosphatemia in patients receiving specialized nutrition support using a graduated dosing scheme: results from a prospective clinical trial.

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OBJECTIVE To determine the safety and efficacy of a graduated dosing scheme of phosphorus replacement therapy in patients with hypophosphatemia receiving specialized nutrition support. METHODS Prospective clinical trial. METHODS A 455-bed tertiary care institution, with Level I trauma

Hypokalemic paralysis in a young obese female.

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BACKGROUND Profound hypokalemia with paralysis usually poses a diagnostic and therapeutic challenge. METHODS We report on a 28-y-old obese Chinese female presenting with sudden onset of flaccid quadriparesis upon awaking in the morning. There is no family history of hyperthyroidism. She experienced

Using phosphate supplementation to reverse hypophosphatemia and phosphate depletion in neurological disease and disturbance.

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Hypophosphatemia (HP) with or without intracellular depletion of inorganic phosphate (Pi) and adenosine triphosphate has been associated with central and peripheral nervous system complications and can be observed in various diseases and conditions related to respiratory alkalosis, alcoholism

Genetic induction of phosphate toxicity significantly reduces the survival of hypercholesterolemic obese mice.

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OBJECTIVE The adverse effects of metabolic disorders in obesity have been extensively studied; however, the pathologic effects of hyperphosphatemia or phosphate toxicity in obesity have not been studied in similar depth and detail, chiefly because such an association is thought to be uncommon.

Hypophosphatemia in patients with essential arterial hypertension.

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The concentration of serum inorganic phosphorus was determined in 162 patients with essential arterial hypertension (EAH) and in 71 control subjects. The serum phosphorus level was lower in patients with essential arterial hypertension than in the control subjects, and the differences were higher in

Acute respiratory failure due to refeeding syndrome and hypophosphatemia induced by hypocaloric enteral nutrition.

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We report a case of acute respiratory failure due to refeeding syndrome caused by hypocaloric enteral tube feeds. A 60-y-old obese man, with a diagnosis of esophageal carcinoma with local metastases, underwent feeding jejunostomy tube insertion. Enteral tube feeding was initiated at small volumes

Hyperphosphatemia is associated with anemia in adults without chronic kidney disease: results from the National Health and Nutrition Examination Survey (NHANES): 2005-2010.

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BACKGROUND Hyperphosphatemia, serum phosphorus ≥ 4.4 mg/dL, is associated with increased risk for chronic kidney disease and cardiovascular disease. Previous studies have shown a weak association between dietary phosphorus intake and serum phosphorus concentrations. While much less common in the

Pathogenesis of hyperostosis: a key role for mesenchymatous cells?

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The similarities between diffuse idiopathic skeletal hyperostosis (DISH) and some forms of ankylosing spondylitis suggest shared pathogenic mechanisms. Entheseal ossification progresses at the same rate in the two conditions, and spondyloarthritis was the first diagnosis considered in several

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