intracranial arteriovenous malformations/adenozină

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Adenosine-induced cardiac standstill to facilitate endovascular embolisation of cerebral arteriovenous malformations in children.

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This report describes three children, aged eight to 11 years, with high-flow cerebral arteriovenous malformations who underwent interventional neuroradiological procedures involving glue (N-butyl cyanoacrylate) embolisation under general anaesthesia. The procedure was facilitated by relative

[Endovascular embolization of high-flow arteriovenous fistulae with non-adhesive agents in the structure of cerebral arteriovenous malformations on the background of adenosine-induced cardioplegia]

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Objective: To analyze the initial results of endovascular embolization of cerebral arteriovenous malformations involving high-flow arteriovenous fistulae with non-adhesive agents on the background of cardioplegia.

Adenosine-induced cardiac pause for endovascular embolization of cerebral arteriovenous malformations: technical case report.

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OBJECTIVE Extremely high flow through arteriovenous malformations (AVMs) may limit the safety and effectiveness of endovascular glue therapy. To achieve a more controlled deposition of glue, we used transient but profound systemic hypotension afforded by an intravenously administered bolus of

Adenosine-induced ventricular asystole to induce transient profound systemic hypotension in patients undergoing endovascular therapy. Dose-response characteristics.

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BACKGROUND Adenosine-induced asystole has been used to induce transient systemic hypotension for various vascular procedures. Dose-response characteristics of adenosine-induced ventricular asystole have not been determined. METHODS During endovascular embolization of cerebral arteriovenous

Effect of adenosine on human cerebral blood flow as determined by positron emission tomography.

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The effect of intravenous infusion of adenosine on CBF was studied in seven patients with cerebral arteriovenous malformation. The patients were examined with positron emission tomography with controlled ventilation using [15O] water and [11C] fluoromethane as tracers. Total and regional CBF were

Neuronal expression of Fas-associated death domain protein and caspase-8 in the perinidal parenchyma of cerebral arteriovenous malformations.

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OBJECTIVE The expression and localization of phosphorylated Fas-associated death domain protein (pFADD) and cleaved caspase-8 was examined in human cerebral arteriovenous malformations (AVMs). The authors focused on the perinidal parenchyma to clarify the effect of AVMs on perinidal brain

Cerebellar arteriovenous malformation with facial paralysis, hearing loss, and tinnitus: a case report.

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OBJECTIVE To describe cerebellar arteriovenous malformation in a 21-year-old man with symptoms resembling those of ear disease and to discuss the relationship between the findings of neurotologic examination and magnetic resonance imaging. METHODS Case report. METHODS Department of Otolaryngology,

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