isoguvacine/ganglion cysts

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Effects of gamma-aminobutyric acid on the compound action potential of the rat superior cervical ganglion.

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The effects on the ganglionic transmission of gamma-aminobutyric acid (GABA) and related drugs were studied in the rat isolated superior cervical ganglion. The extracellularly recorded postganglionic compound action potential was used as an index for ganglionic transmission. GABA reversibly

Retinal GABA neuron labelling with [3H]isoguvacine in different species.

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Retinas from goldfish, chicken, rate, guinea-pig, rabbit and humans were exposed to [3H]isoguvacine either by intravitreal injection in vivo or by incubation in a balanced salt solution. The distribution of radioactivity was then studied by autoradiography. The substance labelled a set of presumed

Isoguvacine, isonipecotic acid, muscimol and N-methyl isoguvacine on the GABA receptor in rat sympathetic ganglia.

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The GABA-mimetic activities of 4 analogues muscimol, isonipecotic acid, isoguvacine and N-methyl isoguvacine have been examined at the GABA receptor in the rat isolated superior cervical ganglion. The depolarizing action of all 4 analogues could be selectively antagonized by bicuculline

Mechanisms of GABA- and glycine-induced increases of cytosolic Ca2+ concentrations in chick embryo ciliary ganglion cells.

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We used fura-2 microfluorometry and the gramicidin-perforated patch clamp technique in an attempt to clarify the mechanisms underlying the GABA- and glycine-induced increases in the cytosolic Ca2+ concentration ([Ca]in) in acutely isolated chick embryo ciliary ganglion neurons. GABA, glycine, and

Type A and B gaba receptors mediate inhibition of acetylcholine release from cholinergic nerve terminals in the superior cervical ganglion of rat.

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The effects of ?-amino-n-butyric acid (GABA), (+)bicuculline, isoguvacine and 3-(4-chlorophenyl)-4-aminobutyrate [(+/-)baclofen] on the K-induced release of [(3)H]acetylcholine (ACh) were studied in the superior cervical ganglia of the rat in vitro. GABA and isoguvacine inhibited [(3)H]ACh release

Effects of glial uptake and desensitization on the activity of gamma-aminobutyric acid (GABA) and its analogs at the cat dorsal root ganglion.

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Responses to gamma-aminobutyric acid (GABA) and 13 structurally related analogs were recorded under voltage clamp conditions from isolated cat dorsal root ganglia (DRG). All of the analogs were applied by superfusion at a concentration of 1 mM. Of the 13 structurally related compounds, only muscimol

Does glial uptake affect GABA responses? AN intracellular study on rat dorsal root ganglion neurones in vitro.

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1. Using single barrel pipettes, intracellular records were obtained from surface neurones of isolated rat dorsal root ganglia (DRG) impaled under microscopic vision.2. Responses to gamma-aminobutyric acid (GABA) were elicited either by ionophoresis or by placing drops of concentrated GABA solutions

GABAA receptor-mediated excitation of nociceptive afferents in the rat isolated spinal cord-tail preparation.

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Algogens such as capsaicin, bradykinin, acetylcholine, 5-hydroxytryptamine and potassium ions applied to exposed tail skin of the rat isolated spinal cord-tail preparation evoke a ventral root response consisting of depolarization and spiking activity. L-glutamate and kainate also evoke similar

Low concentrations of GABA reduce accommodation in primary afferent neurons by an action at GABAB receptors.

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The pattern of accommodation of spike activity during sustained membrane depolarization was investigated in primary afferent neurons recorded intracellularly in vitro in the rat. We show that gamma-aminobutyric acid (GABA) and baclofen reduce accommodation in some fast conducting dorsal root

The role of GABA(A) receptors in the control of transient lower oesophageal sphincter relaxations in the dog.

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OBJECTIVE Transient lower oesophageal sphincter relaxations (TLESRs) are triggered by activation of mechanosensitive gastric vagal afferents and are the major cause of gastroesophageal reflux and therefore an important target for therapeutic intervention in gastroesophageal reflux disease (GERD).

Functional GABAA receptors on rat vagal afferent neurones.

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1. In the present study, in vitro electrophysiology and receptor autoradiography were used to determine whether rat vagal afferent neurones possess gamma-aminobutyric acid (GABA)A receptors. 2. GABA (1-100 microM) and isoguvacine (3-100 microM) caused a concentration-dependent depolarization of the

Autoradiographic distribution of radioactivity from (14)C-GABA in the mouse.

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We investigated the distribution of radioactivity from (14)C-labeled gamma-aminobutyric acid (GABA) in the mouse by in vivo autoradiography to clarify the tissues that show GABA uptake and/or GABA binding. Male mice were injected intravenously with (14)C-GABA in both the absence and presence of an

Coexistence of GABAA and GABAB receptors on A delta and C primary afferents.

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Intracellular recordings from adult rat dorsal root ganglion neurones were performed in vitro and the coexistence of two gamma-aminobutyric acid (GABA) receptors on the membrane of identified A delta and C primary afferents was demonstrated. Transient applications of GABA (10(-6)-10(-2) M) evoked

Inhibitory effect of baclofen on GABA-induced depolarization and GABA-activated current in primary sensory neurons.

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It has been established that GABAA and GABAB receptors can exist separately and/or co-exist in the membrane of dorsal root ganglion neurons. In our previous investigation it has been shown that co-existence of these two kinds of receptors is about 80% of the neurons examined (20/25). The present

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