livedo reticularis/atrofiere

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Cerebrovascular lesions and livedo reticularis (Sneddon's syndrome)--a progressive cerebrovascular disorder?

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Four cases are described in which livedo reticularis was associated with repeated cerebrovascular accidents, which eventually resulted in severe disability in two cases. Patients with severe disability had a history of many years, whereas two patients with little or moderate residual disability had

Superficial Siderosis and Microbleed Restricted in Cortex Might Be Correlated to Atrophy and Cognitive Decline in Sneddon's Syndrome

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Objective: Sneddon's syndrome is a rare non-inflammatory arteriopathy affecting small and medium-sized arteries, characterized by a generalized livedo reticularis and recurrent transient ischemic attack or ischemic stroke. Hemorrhagic stroke was reported in limited cases, but microbleeds and

[Livedo reticularis, acral necroses and renal failure. Cholesterol crystal embolisms].

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In a 67 year old patient with multiple cardiovascular risk factors a livedo reticularis, ischemic acral lesions as well as deterioration of renal function five weeks after cardiac catheterism and aorto-coronary bypass surgery led to suspicion of cholesterol embolism. Fundoscopy revealed cholesterol

[Cobb syndrome and Klippel-Trenaunay-Weber syndrome].

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We reported two patients with neurocutaneous angiomatosis. Patient 1, a 36-year-old woman of Cobb syndrome had cutaneous portwine angiomas in the high back and spinal arteriovenous malformations at the level of Th4-11. She had also atrophy and livedo reticularis presenting during standing position

Systemic lupus erythematosus with unusual maculopapular and erosive cutaneous lesions.

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In 2002, a 38-year-old woman was referred to the department of dermatological sciences for the evaluation of erythematous-infiltrated lesions with prominent purpuric and erosive components distributed on her face, upper chest, and extensor surfaces of the limbs, which developed over a 3-month

Intracerebral hemorrhage associated with Sneddon's syndrome: is ischemia-related angiogenesis the cause? Case report and review of the literature.

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Sneddon's syndrome is characterized by livedo reticularis and multiple ischemic infarcts often associated with antiphospholipid antibodies. Intracerebral hemorrhage (ICH) is unusual in Sneddon's syndrome and has not been reported as the presenting complaint. We report a 38-year-old woman with a

[Neuropathology associated with dementia].

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As dementing diseases are too numerous to refer to all of them, I confine my description to the neuropathology of amyotrophic lateral sclerosis with dementia (ALSD), and cerebral vascular pathology of three unique vascular diseases causing dementia. 1) ALSD: The cortical neuropathology of this

[Cerebrovascular disorders in young patients with livedo racemosa generalisata (Ehrmann)].

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The results of examinations on 15 patients suffering from livedo racemosa generalisata (idiopathic livedo reticularis) are described. 12 of the patients were found to have cerebrovascular disturbances (in most cases severe) and 2 of them had migraine accompagnée. The remaining patient had no

Clinical, neurovascular and neuropathological features in Sneddon's syndrome.

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Sneddon's syndrome (SS) is characterized by ischemic cerebrovascular episodes and livedo reticularis. It is more common in young women and can also be associated with valvulopathy, a history of spontaneous abortion, renal involvement and vascular dementia. We describe three cases of young women with

[Retinal, muscular and cutaneous cholesterol emboli. Progressive encephalopathy].

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A 73 year-old man experienced left monocular blindness and transient right hand clumsiness. A left carotid arteriogram was performed 4 days after admission. Immediately following arteriography, there was a right hemiparesia and dysphasia. After 24 hours, the abnormalities resolved. The patient was

Renal cholesterol embolic disease effectively treated with steroid pulse therapy.

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A 65-year-old man developed acute renal failure with eosinophilia two weeks after a coronary bypass operation and angiography. Renal biopsy revealed cholesterol crystal embolism (CCE) in glomeruli and arterioles. Low-dose corticosteroid therapy failed to recover the renal function; further

Multiple spontaneous small bowel perforations due to systemic cholesterol atheromatous embolism.

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A-65-year-old man was admitted for coronary and peripheral angiography to evaluate angina pectoris and peripheral vascular disease. Following angiography, he suffered from blue toes, livedo reticularis and progressive renal failure. The patient's condition continued to deteriorate, including the

Familial Sneddon's syndrome.

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We report the familial occurrence and apparent autosomal dominant inheritance of Sneddon's syndrome with variable clinical expression. The proband, a 40-year-old woman, presented with livedo reticularis and progressive neurological deterioration following a stroke. The diagnosis was confirmed by

Recurrent spontaneous pneumoperitoneum: A surgical dilemma.

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BACKGROUND Spontaneous pneumoperitoneum describes free air within the peritoneal cavity in the absence of iatrogenic causes or a perforated viscus. This report describes a rare case in which despite raised inflammatory markers, a trial of conservative management proved adequate. METHODS AM is a

Sneddon's syndrome: case report and review of its relationship with antiphospholipid syndrome.

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The Sneddon's syndrome is a rare disorder characterized by the occurrence of cerebrovascular disease associated with livedo reticularis. The antiphospholipid syndrome is the most frequent type of acquired thrombophilia, defined by the occurrence of thrombosis or pregnancy morbidity in the presence

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