noradrenalin/obezitate

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[Study of urinary elimination of aldosterone and catecholamines (adrenalin and noradrenalin) in the syndrome of plethoric obesity].

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Melatonin circadian rhythm in anorexia nervosa and obesity.

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The mean 24-hour secretion and circadian rhythm of melatonin were studied in 12 female subjects with anorexia nervosa (AN), 13 massively obese (OB) women, and 9 normal weight healthy volunteers to investigate the relationship between type of feeding behavior and hormonal secretory pattern. Blood

[Drug therapy of obesity].

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This review is dealing with currently available medications used in the treatment of obesity. Unfortunately currently available drugs did not prove effective in the long term treatment of obese patients. Fenfluramin and D-Fenfluramin has been withdrawn from the market because of severe side effects

New approaches in the pharmacological treatment of obesity.

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Many new substances are currently being investigated for their usefulness in the pharmaco-therapy of obesity. Most drugs interfere with monoamine neuro-transmitter (serotonin, noradrenalin, dopamine and histamine) effects and act as an appetite suppressant. Other approaches are to primarily increase

Obesity-linked homologues TfAP-2 and Twz establish meal frequency in Drosophila melanogaster.

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In all animals managing the size of individual meals and frequency of feeding is crucial for metabolic homeostasis. In the current study we demonstrate that the noradrenalin analogue octopamine and the cholecystokinin (CCK) homologue Drosulfakinin (Dsk) function downstream of TfAP-2 and Tiwaz (Twz)

Over-expression of TLR4-CD14, pro-inflammatory cytokines, metabolic markers and NEFAs in obese non-diabetic Mexicans.

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BACKGROUND Obesity is the world's most important public health problem. Adipose tissue contributes significantly to increase pro-inflammatory mediators whose cascade begins with the union of TLR4 to its microbial ligands (TLR: Toll Like Receptors). It has been reported recently that NEFAs

alpha2-adrenoceptor regulation of the hypothalamic-pituitary-adrenocortical axis in obesity.

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BACKGROUND Abdominal obesity is associated with hyper-responsiveness of the hypothalamic-pituitary-adrenocortical (HPA) axis to stimulatory neuropeptides and to stress. Catecholamines are involved in the regulation of the HPA axis, particularly during stress, via alpha-adrenoceptor

[Daily catecholamine excretion in children suffering from constitutional, exogenous obesity].

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The children from 7 to 15 years of age suffering from constitutional-exogenous overweight , stage I-II, demonstrated a statistically significant increase in adrenaline excretion and a clear tendency to an increase in noradrenaline excretion with the urine. The complex of therapeutic measures

Treatment options for children with monogenic forms of obesity.

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Mutations in genes involved in energy balance regulation within the central nervous system lead to monogenic forms of obesities. Individuals with these mutations are characterized by early-onset obesity and in some cases by endocrine abnormalities. Carriers of leptin gene mutations are able to

Duloxetine in obese binge eater outpatients: preliminary results from a 12-week open trial.

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OBJECTIVE Attempts have been made to find appropriate drug regimens to treat binge eating disorder (BED). Several reports have examined the use of selective serotonin reuptake inhibitors (SSRIs) or mood stabilizers; both serotonin and noradrenalin reuptake inhibitors (SNRIs) have been reported to be

Usefulness of combining intermittent hypoxia and physical exercise in the treatment of obesity.

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Obesity is an important public health problem worldwide and is a major risk factor for a number of chronic diseases such as type II diabetes, adverse cardiovascular events and metabolic syndrome-related features. Different treatments have been applied to tackle body fat accumulation and its

Resting metabolic rate, fat-free mass and catecholamine excretion during weight loss in female obese patients.

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The reduction in resting metabolic rate (RMR) during weight loss exceeds that accounted for by changes in body composition by 15%, suggesting that factors other than fat-free mass (FFM) explain the metabolic adaptation during food restriction in obesity. Our study aimed to establish if changes in

[Effect of thyroid hormones on noradrenaline-stimulated lipolysis in obesity].

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11 extremely adipose patients were over 4 days loaded with a noradrenalin infusion of 0.1 microgram/kg body weight/minute over 60 minutes before and after a daily application of 200 micrograms tri-iodothyronine. The determination of the free fatty acids and of glycerin resulted in a significant

Enhancement of lipolytic responsiveness of adipocytes by novel plant extract in rat.

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Subcutaneous adipocytes accumulate excess energy as triglycerides, but lipolytic response is less sensitive to catecholamines than visceral adipocytes. Obesity also induces catecholamine resistance of adipocytes. We have searched for crude drugs that could enhance the lipolytic response to

[Weight loss via drug therapy].

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Obesity and its associated diseases are an increasing challenge in medicine. A change in lifestyle is usually the first step with modifications in nutrition, physical activity and behavior. However, most of obese patients are not able to follow such a treatment regimen for a longer period of time.

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