phantom limb/inflamație

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Supernumerary phantom limbs in ICU patients with acute inflammatory demyelinating polyneuropathy.

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OBJECTIVE To explore the previously undescribed phenomenon of phantom limb generation in patients with severe acute inflammatory demyelinating polyradiculoneuropathy (AIDP). METHODS Between April 2011 and January 2014, we encountered 3 patients with AIDP in our intensive care unit who experienced

Self-reported treatments used for lower-limb phantom pain: descriptive findings.

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OBJECTIVE To examine the types and relative frequency of treatments used for lower-limb phantom limb pain (PLP), including both pharmacologic and rehabilitative interventions. METHODS Cross-sectional survey. METHODS Community-based survey from clinical databases. METHODS Community-based sample

The impact of immunological parameters on the development of phantom pain after major amputation.

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OBJECTIVE To investigate the relationship between local and systemic inflammatory markers and phantom limb pain. METHODS In 39 consecutive patients undergoing major amputations nerve biopsies, serum and clinical data was collected. Patients were followed up for 12 months to report on the incidence

Joint stiffness in a phantom limb: evidence of central nervous system involvement in rheumatoid arthritis.

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OBJECTIVE The nature and cause of perceived joint stiffness (PJS), a well-established and defining symptom of rheumatoid arthritis (RA), remains unclear. We hypothesized that changes in the central nervous system (CNS) may determine and maintain this subjective experience of stiffness in a limb even

[The effect of fenazepam and sidnocarb in the phantom pain syndrome].

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The phantom-pain syndrome model was used to examine the effects of phenazepam, sydnocarb and their combination in chronic oral administration. Phenazepam was shown to have no effects on the development of the phantom-pain syndrome. Sydnocarb arrested the progression of the pain syndrome, reduced its

Factors affecting phantom limb pain in patients undergoing amputation: retrospective study.

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The efficacy of preemptive analgesia for prevention of phantom limb pain has been controversial although pain management before amputation is empirically important. The aim of this study was to determine the associated factors with perioperative phantom limb

Profound pain reduction after induction of memantine treatment in two patients with severe phantom limb pain.

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We present the cases of two patients who suffered severe lower extremity injuries and subsequently developed phantom limb pain (PLP) that was refractory to high dose opioids and adjunctive pain medications. Both patients were receiving large doses of oral methadone, IV hydromorphone via a

Pharmacological interventions for phantom limb pain.

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OBJECTIVE To review the mechanisms and current clinical application of pharmacological interventions for phantom limb pain. METHODS Both Chinese and English language literatures were searched using MEDLINE (1982 - 2011), Pubmed (1982 - 2011) and the Index of Chinese Language Literature (1982 -

Phantom pain and sensation among British veteran amputees.

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Using a mail-delivered questionnaire, we surveyed 590 veteran amputees concerning phantom pain, phantom sensation and stump pain. They were selected randomly from a population of 2974 veterans with long-standing limb amputation(s) using a computer random number generator. Eighty-nine percent

[Neural basis of pain].

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Main elements concerning the physiology of pain are described, as well as the structures of the nervous system at the origin of the central control of pain: peripheral fibres (small diameter myelinated A delta and unmyelinated C fibres); spinal ascending pathways; cerebral structures relaying

Alteration of neuronal activity after digit amputation in rat anterior cingulate cortex.

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Phantom limb pain is experienced by nearly 50 - 80% of the patients following limb amputation. The anterior cingulate cortex (ACC) is a part of the limbic system that is an essential component in mediating the affective and emotional component of pain responses. To explore the role of ACC in the

Epithelial growth factor receptor (EGFR)-inhibition for relief of neuropathic pain-A case series.

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Background Neuropathic pain remains a significant challenge with unsatisfactory therapeutic options. Its pathogenesis may involve the neuropathic triad of neuronal, glial and immune cells. Communication between these cells is possibly perpetuated by mitogen-activated protein kinase (MAPK)-signaling.

Preventing the development of chronic pain after orthopaedic surgery with preventive multimodal analgesic techniques.

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The prevalences of complex regional pain syndrome, phantom limb pain, chronic donor-site pain, and persistent pain following total joint arthroplasty are alarmingly high. Central nervous system plasticity that occurs in response to tissue injury may contribute to the development of persistent

Development of a Multimodal Analgesia Protocol for Perioperative Acute Pain Management for Lower Limb Amputation.

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Multimodal analgesia may include pharmacological components such as regional anesthesia, opioid and nonopioid systemic analgesics, nonsteroidal anti-inflammatories, and a variety of adjuvant agents. Multimodal analgesia has been reported for a variety of surgical procedures but not yet for lower

[Pharmacotherapy for neuropathic pain caused by injury to the afferent nerve fibers].

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Phantom pain, a form of neuropathic pain, is caused by damage to somatosensible afferent nerve fibres in the peripheral or central nervous system. Often, the pain cannot be satisfactorily treated with nonsteroidal anti-inflammatory drugs. Dependent on the underlying mechanism the pain is treated

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