polycythemia vera/hypoxia

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Consequences of moderate hypoxia on red cell glycolytic metabolism in polycythemia rubra vera.

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Red blood cell metabolism was studied in male patients with polycythemia rubra vera (Vaquez disease) and compared to a reference sample of 74 healthy adults. A total of seventeen enzyme activities and seventeen metabolite concentrations were determined. In addition to the modifications of the

Could hypoxia increase the prevalence of thrombotic complications in polycythemia vera?

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Thromboses represent a major cause of morbidity and mortality in polycythemia vera but the contributing mechanisms are not fully described. To evaluate whether environmental conditions such as altitude/hypoxia could impact thrombosis history, we retrospectively analyzed thrombosis history in 71

Sever hypoxemia in an obses patient with polycythemia vera.

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Studies on the response of hypoxia and relative hyperoxia in two polycythemia vera patients.

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The mouse with hypoxia-induced erythremia, an erythropoietin bioassay animal.

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HYPOXIA IN POLYCYTHEMIA VERA.

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Sensomotor axonal peripheral neuropathy as a first complication of polycythemia rubra vera: A report of 3 cases.

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METHODS Female, 64 FINAL DIAGNOSIS: Polycythemia rubra vera Symptoms: Burning pain • cramps • hypesthesia • itching • paresthesia METHODS - Clinical Procedure: - Specialty: Neurology. OBJECTIVE Unusual clinical course. BACKGROUND The association between polycythemia vera and peripheral neuropathy

[Leg ulcers in patient affected by polycythemia vera in treatment with hydroxycarbamide. Case report].

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Leg ulcers are a frequent and serious complication of polycythemia vera (PV). They are caused by a synergic action of vascular, neurological and infectious physiopathologic mechanisms. Furthermore, cytostatic therapies commonly employed to control the myeloproliferative disease negatively interfere

Incidence of polycythemia vera in a defined population.

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In this retrospective investigation from Malmö, a city well-suited for epidemiologic studies, 177 patients (88 males and 89 females) with polycythemia vera (PV) were identified between 1950 and 1984. The incidence rate (number of cases/100,000/yr) in both sexes increased significantly, being 1.0 in

Recent advances in the molecular biology of congenital polycythemias and polycythemia vera.

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This review will focus on the molecular basis of certain polycythemic disorders. Primary polycythemias are characterized by acquired somatic or inherited germ-line mutations expressed within hematopoietic progenitors that cause increased accumulation of red blood cells. Polycythemia vera (PV), an

A 38-Year-Old Man With Well Treated OSA on CPAP With Persistent Nocturnal Hypoxemia.

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A 38-year-old male with a prior diagnosis of severe OSA (apnea-hypopnea index [AHI] 99/h) presented for transfer of care. He was successfully titrated to CPAP of 10 cm H₂O at an outside laboratory and was compliant with therapy with residual AHI 1.9/h. On presentation, he was

[Leg ulcer in a patient treated with hydroxyurea for polycythemia vera].

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BACKGROUND Causes of leg ulcers vary widely, although venous insufficiency and peripheral arteriopathy are most common. Ulcers are much rarer in patients treated by hydroxyurea. METHODS A 66-year-old woman who had been treated with hydroxyurea for polycythemia vera for four years came to our

Polycystic kidney disease and polycythemia vera. Occurrence in a patient receiving hemodialysis.

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Symptomatic erythrocytosis developed in a 59-year-old man with polycystic kidney disease (PKD) while he was receiving maintenance hemodialysis. Major clinical and laboratory data suggested a diagnosis of polycythemia vera (PV), despite a normal serum alkaline phosphatase level and leukocyte count.

Polycythemia vera.

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Polycythemia vera (PV) is one of the myeloproliferative diseases, and, as such, is an example of clonal hematopoiesis. The progeny of a single, abnormal, hematopoietic stem cell gain a growth advantage over their normal counterparts resulting in overproduction of red cells generally accompanied by

Evaluation of diagnostic criteria in polycythemia vera.

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There is no single diagnostic marker for the only known type of primary acquired erythrocytosis, polycythemia vera (PV). The Polycythemia Vera Study Group (PVSG) used a combination of major and minor diagnostic criteria. However, these guidelines have some limitations and in the presence of newer

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