pterygium/hypoxia

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TNF-α upregulates HIF-1α expression in pterygium fibroblasts and enhances their susceptibility to VEGF independent of hypoxia.

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The clinical manifestations of pterygium are characterized by rapid growth and postoperative recurrences. We had previously proposed that hypoxia-inducible factor (HIF)-1α recruits progenitor cells during the development and progression of pterygia. Recently, it was reported that various stimuli,

Capillaries in the epithelium of pterygium.

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OBJECTIVE To present new morphological observations of intraepithelial capillaries in pterygium and to provide some explanations for this phenomenon. METHODS The ultrastructural features of pterygia from 26 patients were examined. Surgically excised tissue was processed for conventional light and

Cultivation and characterization of pterygium as an ex vivo study model for disease and therapy.

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OBJECTIVE Development of ex vivo model to study pathogenesis, inflammation and treatment modalities for pterygium. METHODS Pterygium obtained from surgery was cultivated (3 months). Gravitational attachment method using viscoelastic facilitated adherence of graft and outgrowing cells. Medium

Ηypoxia-inducible factor-1α, von Hippel-Lindau protein, and heat shock protein expression in ophthalmic pterygium and normal conjunctiva.

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OBJECTIVE Εnhanced expression of transcription factor hypoxia inducible factor HIF-1α is known to play a critical role in the modulation of cell metabolism and survival pathways as well as having stem-cell-like properties. Furthermore, accumulated data reveal the existence of cross-regulation

Ischemic tissue injury and progenitor cell tropism: significant contributors to the pathogenesis of pterygium.

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Pterygium is a common ocular surface disease characterized by triangular wing-like growth consisting of subconjunctival hypertrophic connective tissue. Pterygium is easily complicated by adhesion to the eyelid and diplopia related to motility restriction of the eyeball. Beyond the cosmetic problems,

Endothelial progenitor cells in pterygium pathogenesis.

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OBJECTIVE The pathogenesis of pterygium is not well known, and controversy exists about the cell origins and the nature of initial trigger required for its development. We investigated whether endothelial progenitor cells (EPCs) are involved in pathogenesis of pterygium and the mechanism underlying

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