purpura fulminans/febră

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Acute infectious purpura fulminans due to probable spotted fever.

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Purpura fulminans (PF) is associated with several infections, most notably with meningococcus, staphylococcus, and streptococcus infections. However, there are few reports of association of this entity with spotted fever from India. We report the case of a 55-year-old man who presented with fever,

Brazilian purpuric fever: epidemic purpura fulminans associated with antecedent purulent conjunctivitis. Brazilian Purpuric Fever Study Group.

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In late 1984, 10 children in a small, rural town in Brazil had high fever associated with vomiting and abdominal pain. Within 12-48 h of the onset of fever, purpura developed associated with vascular collapse and peripheral necrosis. All 10 children died. Cerebrospinal fluid examinations did not

Two Cases of Israeli Spotted Fever with Purpura Fulminans, Sharon District, Israel.

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We report a series of 5 case-patients who had Israeli spotted fever, of whom 2 had purpura fulminans and died. Four case-patients were given a diagnosis on the basis of PCR of skin biopsy specimens 3-4 days after treatment with doxycycline; 1 case-patient was given a diagnosis on the basis of

Idiopathic purpura fulminans in dengue hemorrhagic fever.

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Purpura fulminans is a rapidly progressive thrombotic disease that has been described during both severe bacterial and viral infections. Disseminated intravascular coagulation (DIC), antiphospholipid antibodies and acquired or congenital C and S protein deficiency are thought to play a role in its

[Purpura fulminans in scarlet fever].

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Purpura fulminans complicating scarlet fever.

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Purpura fulminans; report of two cases following scarlet fever.

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PURPURA FULMINANS FOLLOWING A MILD CASE OF SCARLET FEVER.

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[A case of post-scarlet fever "purpura fulminans" with hyperfibrinolysis treated with epsilon-aminocaproic acid].

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Fatal purpura fulminans and Waterhouse-Friderichsen syndrome from fulminant Streptococcus pneumoniae sepsis in an asplenic young adult.

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Asplenic patients are at increased risk for sepsis and fulminant infection. Sepsis in these patients is typically secondary to encapsulated bacteria, with Streptococcus pneumoniae being the most frequent pathogen. Rare complications of severe sepsis include purpura fulminans and bilateral adrenal

[Purpura fulminans (symmetric peripheral gangrene): 7-year consecutive case review in Japan].

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BACKGROUND Acute infectious purpura fulminans (AIPF), a rare syndrome of infection-induced ischemic necrosis of the extremities (symmetric peripheral gangrene), is due most often to Neisseria meningitidis in the US and the UK, but is not endemic to Japan. OBJECTIVE We reviewed clinical AIPF

Purpura fulminans in a complicated Falciparum malaria.

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A 19-year-old male presented with fever, oliguria and purpuric lesions involving both hands. The patient was diagnosed as a case of purpura fulminans with disseminated intravascular coagulation due to complicated Falciparum malaria. The case is presented to sensitize the physicians to keep malaria

Purpura fulminans secondary to Xanthomonas maltophilia sepsis in an adult with aplastic anemia.

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Purpura fulminans is a rare disease characterized by purpura ecchymosis, hypotension, and fever associated with disseminated intravascular coagulation. It often begins as a benign infectious process and subsequently progresses to a severe, catastrophic outcome. It is recognized to originate from

Neonatal purpura fulminans manifestation in early-onset group B Streptococcal infection.

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METHODS Male, 0 FINAL DIAGNOSIS: Purpura fulminans Symptoms: Fever • letargy METHODS - Clinical Procedure: - Specialty: Pediatrics and Neonatology. OBJECTIVE Rare disease. BACKGROUND Neonatal purpura fulminans (PF) is a rare but frequently fatal disorder associated with high morbidity and mortality.

[A 52-year-old woman with acute shock and purpura fulminans. Pneumococcal sepsis].

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We report a 52-year-old female patient admitted with fever, chills, and myalgias since the previous day. On the day of admission she had a generalized seizure. The patient had no previous illnesses. Laboratory investigations showed consumptive coagulopathy with clinical manifestations of shock and

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