stomach diseases/edema

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[Hypoproteinemia of high grade and anasarca caused by chronic gastropathy with giant hypertrophy of the mucosa and loss of plasmatic proteins into the gastric lumen].

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Differential diagnosis of reactive gastropathy.

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Reactive or chemical gastropathy is the constellation of endoscopic and histological changes caused by chemical injury to the gastric mucosa. Its diagnosis rests on the histopathological demonstration of nonspecific elementary lesions that may occur simultaneously or separately in different degrees

[Gastritis and gastropathies].

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A review of the different types of gastritis, including features in which the edema and erythema can resemble the picture, but without the typical inflammatory reaction that is called gastropathy. A description of various epidemiological, aetiological, pathogenetic, are proposed. The review

Portal gastropathy and duodenopathy in children with extrahepatic and intrahepatic portal hypertension: endoscopic diagnosis and histologic scoring.

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OBJECTIVE The aim of the study was to determine the frequency of portal gastropathy (PG) and duodenopathy (PD) in children, to document the correlation of various clinical and laboratory parameters associated with portal hypertensive gastroduodenal lesions, to compare the endoscopic portal

Hemorrhagic gastropathy in epidemic nephropathy.

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A patient with epidemic nephropathy (NE) and with gastrointestinal symptoms and hemorrhagic gastropathy prompted us to study further 10 consecutive patients with NE. Gastroscopy was carried out within 1 to 4 weeks after the beginning of the symptoms, and in every case a hemorrhagic gastropathy was

[Modified Cr51-chromium-albumin test for the differential diagnosis of exudative gastropathies and enteropathies].

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A modification of the 51Cr-chromium-albumin test allows differentiation between protein-losing gastropathy and protein-losing enteropathy. After i.v. application of 51Cr-chromium-albumin, radioactivity is measured in the gastric fluid (basal after pentagastrin and after atropine, each for 1 h) and

Hyperplastic gastropathy. Clinicopathologic correlation.

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Assessment of gastric histology in patients with hyperplastic gastritis is now possible using new endoscopic biopsy techniques that obtain a full-thickness gastric mucosal biopsy. Thirty-one patients with hyperplastic gastropathy and full-thickness gastric mucosal biopsies or gastric resections were

Uremic gastropathy in the dog.

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Stomachs of four dogs with uremia and four normal dogs were examined. Uremic stomachs represented four types of disease: atrophic, amyloidotic, ulcerative and necrotic gastropathy. Pathologic changes common to all uremic stomachs were expansion of the lamina propria, atrophy of gastric glands, and

Benign hypertrophic gastropathy: ménétrier's disease in childhood and adolescence.

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Menetrier's disease (hypertrophic gastropathy) in patients under the age of 20 years is rare. A case is described and the features of the condition found in 11 young patients are discussed. The patients, when seen, are usually in acute abdominal pain and vomiting or edema due to hypoproteinemia is

[The role of cyclooxygenase 2 and prostaglandin I2 in the development of portal hypertensive gastropathy].

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OBJECTIVE To study the role of cyclooxygenase 2 (COX 2) and prostaglandin I2 (PGI2) in the development of portal hypertensive gastropathy (PHG). METHODS Forty Wistar rats were divided into surgery group (32) and control group (8). Partial portal vein ligation method was used to narrow the sectional

Treatment of protein-losing gastropathy with atropine.

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Protein loss from the gastric mucosa with hypertrophic gastric folds and hypoalbuminemia has been associated with low, normal and elevated gastric acid output. A case of protein-losing gastropathy with slightly elevated gastric acid output is described. Associated findings were hypertrophic gastric

Relationship among serum creatinine, serum gastrin, calcium-phosphorus product, and uremic gastropathy in cats with chronic kidney disease.

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BACKGROUND Chronic kidney disease (CKD) in cats is associated with gastrointestinal signs commonly attributed to uremic gastropathy. Consequently, patients often are treated with antacids and gastrointestinal protectants. This therapeutic regimen is based on documented gastric lesions in uremic

New frontiers in gut nutrient sensor research: prophylactic effect of glutamine against Helicobacter pylori-induced gastric diseases in Mongolian gerbils.

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Ammonia is one of the important toxins produced by Helicobacter pylori (H. pylori), the major cause of peptic ulcer diseases. We examined whether glutamine or marzulene (a gastroprotective drug containing 1% sodium azulene and 99% glutamine) protects the gastric mucosa against H. pylori in vivo and

Protein-Losing Enteropathy and Gastropathy.

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The diagnosis of protein-losing enteropathy (PLE) should be considered in all patients with hypoalbuminemia and edema without other known causes, and established by plasma alpha(1)-antitrypsin (alpha(1)-AT) clearance or nuclear studies. The therapy for PLE should focus principally on the treatment

Ampullary carcinoma associated with protein-losing gastropathy due to diffuse varioliform gastritis.

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Protein-losing gastropathy due to diffuse varioliform gastritis is a rare condition, and its occurrence accompanying ampullary carcinoma is particularly rare. We report here a case of ampullary carcinoma accompanied with protein-losing gastroenteropathy due to diffuse varioliform gastritis. A

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