toxemia/triglyceride

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Triglycerides and apoproteins in toxemia of pregnancy.

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In order to clarify the mechanism of hypertriglyceridemia caused by toxemia of pregnancy, apoprotein B, CII, CIII, and E in serum were determined by single radial immunodiffusion, and apoprotein CIII0, CII, CIII1 and CIII2 in very low density lipoprotein (VLDL) fractionated by ultracentrifugation

[Changes in serum lipoprotein (a) levels related to hyperlipidemia during pregnancy--comparing normal pregnancy and toxemia of pregnancy].

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In this study, we investigated fluctuations in serum lipoprotein (a) (Lp(a)) levels in normal and toxemic pregnancy. We measured serum total cholesterol (TC), triglyceride (TG), phospholipid (PL), high-density lipoprotein (HDL), apolipoprotein and Lp(a) levels in 33 normal pregnant and 11 toxemic

Studies on lipoperoxide of normal pregnant women and of patients with toxemia of pregnancy.

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1. Serum lipoperoxide was determined by the method of Yagi et al. (Yagi, K., Nishigaki, I. and Ohama, H. (1968) Jap. J. Vitamin 37, 105) for 22 cases of non-pregnant women, 24 cases of 1st trimester of pregnancy (1-12 weeks), 39 cases of 2nd trimester of pregnancy (13-26 weeks), 58 cases of 3rd

Metabolic profiles in goat does in late pregnancy with and without subclinical pregnancy toxemia.

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BACKGROUND Pregnant goat does can develop various metabolic diseases during late pregnancy that may have profound effects on their health and productivity, including subclinical pregnancy toxemia. OBJECTIVE This study was performed to evaluate serum biochemical findings in goats in late pregnancy

Molecular mechanisms of lipid metabolism disorder in livers of ewes with pregnancy toxemia.

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Pathogenesis of pregnancy toxemia (PT) is believed to be associated with the disruption of lipid metabolism. The present study aimed to explore the underlying mechanisms of lipid metabolism disorder in the livers of ewes with PT. In total, 10 pregnant ewes were fed normally (control group) whereas

Brief overview of maternal triglycerides as a risk factor for pre-eclampsia.

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BACKGROUND Features of the metabolic syndrome-maternal obesity, diabetes mellitus and chronic hypertension-are risk factors for pre-eclampsia. OBJECTIVE To determine the risk of pre-eclampsia in the presence of maternal hypertriglyceridemia, another major element of the metabolic

Cord blood hypertriglyceridemia in Chinese newborns: an index of fetal distress.

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Cord serum triglyceride concentrations in 214 Chinese newborns were measured at birth with the Hitachi Model 716 autoanalyzer using the G-3-PDH UV end point method. The mean and the 95th percentile value in a normal population of 100 newborns were found to be 28 mg/100 ml and 48 mg/100 ml,

[Changes in lipid metabolism in pregnancy in relation to gestoses].

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In 21 healthy pregnant women and 21 women with toxemias in pregnancy between 34 and 36 gestational weeks were the following parameters lipid metabolism have been analyzed: triglyceride, cholesterol, HDL cholesterol, LDL cholesterol. The lipid data of the healthy pregnant women did not differ

Plasma concentrations of key metabolites and insulin in late-pregnant ewes carrying 1 to 5 fetuses.

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Ewes bearing more than 1 fetus are more susceptible to pregnancy toxemia than those with a single fetus. Crossbreeding programs in Israel increased the occurrences of ewes bearing more than 2 fetuses; therefore, the aim was to assess the exacerbation in the metabolic status of ewes pregnant with

[Maternal nutritional states and serum insulin-like growth factor-I (IGF-I) concentrations in normal and abnormal pregnancy].

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It is well known that serum IGF-I concentrations are regulated endocrinologically since IGF-I has a growth-promoting action as a mediator of growth hormone. However, recent reports suggest that nutritional states influence serum IGF-I concentration because IGF-I shows anabolic effects like insulin.

Summary of the NATO advanced research workshop on dietary omega 3 and omega 6 fatty acids: biological effects and nutritional essentiality.

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A number of human studies presented at the workshop indicate that the premature infant at birth is biochemically deficient in docosahexaenoic acid (DHA) in both the brain and liver phospholipids, and that DHA is essential for normal visual acuity. The amount of DHA necessary to maintain normal

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