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bruxism/дофамин

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Страница 1 от 37 полученные результаты

A randomised, open-label, crossover study of the dopamine agonist, pramipexole, in patients with sleep bruxism.

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Sleep bruxism bears several similarities to restless legs syndrome, and a link to changes in central dopamine activity has been considered in both conditions. The dopamine agonist pramipexole is currently indicated for the symptomatic treatment of restless legs. The effect of pramipexole on sleep

Single nucleotide polymorphisms in dopamine receptor D2 are associated with bruxism and its circadian phenotypes in children.

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Objective: To evaluate the association of bruxism phenotypes with single nucleotide polymorphisms in FKBP5, DRD2, ANKK1, and COMT.Methods: Clinical oral examination was performed to diagnose bruxism phenotypes in 150 children. DNA was collected from saliva. Logistic

Drugs and bruxism: a critical review.

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OBJECTIVE Bruxism associated with drugs can be destructive, resulting in severe consequences to health that include destruction of tooth structure, irreversible harm to the temporomandibular joint, severe myofascial pain, and muscle contraction headache. However, reports concerning a possible

[Bruxism as a side effect of serotonin re-uptake inhibitors].

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The Netherlands Pharmacovigilance Centre Lareb received 14 reports on drugs which had teeth grinding as a side effect. The majority of reports (12) concerned selective serotonin re-uptake inhibitors. Several case reports in the literature support the association between using selective serotonin

Reevaluating Antidepressant Selection in Patients With Bruxism and Temporomandibular Joint Disorder.

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Temporomandibular joint disorder (TMD) is a broad pain disorder that refers to several conditions affecting the temporomandibular joint of the jaw and the muscles of mastication. As with most pain disorders, a high prevalence of depression and anxiety is associated with TMD. Research has shown that

Possible paroxetine-induced bruxism.

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OBJECTIVE To report the case of a patient with possible paroxetine-induced bruxism that was effectively treated with buspirone. METHODS A 20-year-old woman with no active medical conditions besides acne and no history of dental problems was seen in an outpatient psychiatry clinic for the evaluation

Buspirone as an antidote to SSRI-induced bruxism in 4 cases.

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BACKGROUND One hypothesis to explain selective serotonin reuptake inhibitor (SSRI)-induced bruxism states that SSRIs increase extrapyramidal serotonin levels, thereby inhibiting dopaminergic pathways controlling movement. Previous reports have emphasized buspirone's postsynaptic dopaminergic effect

Evaluation of the effectiveness of infrared light-emitting diode photobiomodulation in children with sleep bruxism: Study protocol for randomized clinical trial.

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Sleep bruxism is a masticatory muscle activity characterized as rhythmic (phasic) or nonrhythmic (tonic). In children and adolescents, etiological factors, such as breathing pattern and sleep quality, have recently been addressed in studies investigating sleep bruxism. New therapies

Ketotifen-induced nocturnal bruxism.

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Nocturnal bruxism is a common oromandibular movement disorder highly prevalent in children, but its pathophysiological mechanism has not been fully explained. Iatrogenic sleep bruxism has been described following treatment with several psychotropic medications. However, no case of

Urinary catecholamine levels and bruxism in children.

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This study was performed to test the hypothesis that emotionally stressful states measured by the urinary catecholamines may affect the development of bruxism. Three hundred and fourteen children, boys and girls, aged 6-8 years were included in this study. Bruxism was recorded by a clinical

Urinary levels of catecholamines among individuals with and without sleep bruxism.

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BACKGROUND Sleep bruxism (SB) is characterized by repetitive and coordinated mandible movements and non-functional teeth contacts during sleep time. Although the etiology of SB is controversial, the literature converges on its multifactorial origin. Occlusal factors, smoking, alcoholism, drug usage,

Effects of pergolide on severe sleep bruxism in a patient experiencing oral implant failure.

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In the patient described in this study, oral implants failed as a probable consequence of severe, polysomnographically confirmed sleep bruxism. As this patient had the wish to be re-implanted after this failure, we decided to try diminishing the frequency of bruxism and duration first. To that end,

Double-blind, crossover, placebo-controlled trial of bromocriptine in patients with sleep bruxism.

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This study was designed to assess the effects of bromocriptine, a dopamine D2 receptor agonist, on sleep bruxism. Seven otherwise healthy patients with severe and frequent sleep bruxism participated in this randomized, double-blind, placebo-controlled study. The study used a crossover design that

Relationship between stress and sleep bruxism in children and their mothers: A case control study.

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Sleep bruxism (SB) is a frequent condition in children. Its pathophysiology involves certain neurotransmitters (serotonin, dopamine, noradrenalin, histamine), but the environment seems to influence its occurrence.The objective of this study was to

Tobacco use and reported bruxism in young adults: a nationwide Finnish Twin Cohort Study.

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BACKGROUND Higher levels of smoking, leading to increased levels of nicotine and dopamine release, may be more strongly related to bruxism, although this relationship has remained unclear. Thus, the aim of the present study was to investigate the possible effect of cumulative tobacco use on bruxism
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