diabetic nephropathies/альбумины

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Amelioration of diabetic nephropathy by treatment with monoclonal antibodies against glycated albumin.

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The pathogenesis of diabetic nephropathy is incompletely understood, but increased nonenzymatic glycation of proteins is considered an important contributory factor. Glycated albumin, which is increased in diabetic sera and is preferentially transported into the renal glomerulus, induces an increase

Urinary albumin excretion is correlated to fibrinogen levels and protein S activity in patients with type 1 diabetes mellitus without overt diabetic nephropathy.

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The aim of the study was to test the hypothesis that in diabetic patients without overt nephropathy there may be a correlation between the activity of natural anticoagulant proteins and glomerular dysfunction. Assays for functional activity of proteins S and C, measurements of urinary albumin

'Microalbuminuria' and diabetes: a critique--assessment of urinary albumin excretion and its role in screening for diabetic nephropathy.

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Abnormal albumin excretion in the range not previously detectable by routine clinical methods can now be readily quantified, and has been shown to predict the development of clinically significant nephropathy in insulin-dependent diabetes mellitus (IDDM) and to predict excess mortality in

Glycation of serum albumin and its role in renal protein excretion and the development of diabetic nephropathy.

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The present study was designed to investigate whether microalbuminuria at the onset of diabetic nephropathy might be partially due to the glycation of serum albumin. It is postulated elsewhere (Ghiggeri et al., Proc. Eur. Dial. Transplant. Assoc. 21 (1984) 633-636) that the glycation of serum

[Albumin-to-creatinine ratio as a diagnostic tool for type 2 diabetic nephropathy].

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BACKGROUND Given the low effectiveness of 24 hours determination of urinary albumin excretion (UAE) for the diagnosis of diabetic nephropathy in primary care, we aimed at evaluating the albumin: creatinine ratio (ACR) in the first voided morning urine sample as a diagnostic tool in this

The validity of random urine specimen albumin measurement as a screening test for diabetic nephropathy.

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To assess the validity of urine albumin concentration (UAC) and the urine albumin:creatine ratio (UACR) in a random urine specimen (RUS) for screening diabetic nephropathy in Korea, a total of 105 ambulatory diabetes mellitus patients (male:female, 52:53), ages 40-75 years (median 59 years)

Effects of the angiotensin II type 1 receptor antagonist candesartan, compared with angiotensin-converting enzyme inhibitors, on the urinary excretion of albumin and type IV collagen in patients with diabetic nephropathy.

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BACKGROUND We previously reported that the angiotensin II type 1 receptor antagonist candesartan was effective in reducing blood pressure and microalbuminuria in hypertensive patients with diabetic nephropathy after angiotensin-converting enzyme (ACE) inhibitors were replaced due to side effects. In

Effects of captopril on urinary excretion of albumin and prostaglandins in patients with diabetic nephropathy.

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Recently the beneficial effects of captopril (angiotensin-converting-enzyme inhibitor) on diabetic nephropathy, especially proteinuria, have been reported by some investigators. The mechanism whereby proteinuria is reduced, however, have not been clarified yet. The present study was undertaken to

Spot urine albumin to creatinine ratio and serum cystatin C are effective for detection of diabetic nephropathy in childhood diabetic patients.

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Spot urinary albumin to creatinine ratio (ACR) measurement has been suggested as a surrogate to 24-hr urine collection for the assessment of microalbuminuria, and cystatin C (cysC) is known as an advantageous marker for renal function. The aim of this study was to evaluate the clinical values of

Urinary albumin fragments as a new clinical parameter for the early detection of diabetic nephropathy.

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Urinary albumin fragments (uAF) from patients with NIDDM were analyzed as a possible factor in the early discovery of diabetic nephropathy before emergence of microalbuminuria. SDS-PAGE and immunoblot assay were employed for detection of uAF. Samples from 252 patients with NIDDM, 158 patients with

Plasma homocysteine is related to albumin excretion rate in patients with diabetes mellitus: a new link between diabetic nephropathy and cardiovascular disease?

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The high risk of cardiovascular disease in patients with diabetes mellitus, particularly in those with nephropathy, is not completely explained by classical risk factors. A high plasma homocysteine concentration is an independent risk factor for cardiovascular disease but information on its

Prolactin receptor mRNA expression in experimental diabetic nephropathy: Relationship with urinary albumin excretion.

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OBJECTIVE Disturbances of prolactin secretion, peptide hormone produced by the pituitary gland, occur in both the chronic renal failure and in diabetes mellitus. So far, the role that prolactin play in the pathology of diabetic nephropathy progression is still unclear. Therefore, the present study

Enhancing the predictive value of urinary albumin for diabetic nephropathy.

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Diabetic nephropathy (DN) is a growing cause of ESRD despite widely known recommendations for improved glycemic and BP control. Perhaps earlier identification of patients who have diabetes and are at high risk for DN could reverse these epidemiologic trends. Albumin excretion rate (AER), the

The endothelial glycocalyx as a key mediator of albumin handling and the development of diabetic nephropathy.

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The endothelial glycocalyx is a complex mesh of proteoglycans, glycoproteins and other soluble components which covers the vascular endothelium. It is considered to play an important role in many physiological processes including vascular permeability, transduction of shear stress and interaction of

Near-normal urinary albumin concentrations predict progression to diabetic nephropathy in Type 1 diabetes mellitus.

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OBJECTIVE To determine urinary albumin concentrations that predict progression to diabetic nephropathy and sight-threatening diabetic retinopathy and identify baseline parameters associated with progression. METHODS One thousand two hundred and one Type 1 diabetic patients aged 35 years or younger

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