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salivary gland neoplasms/phosphatase
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Страница 1 от 16 полученные результаты
Prostate marker immunoreactivity in salivary gland neoplasms. A rare pitfall in immunohistochemistry.
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A 66-year-old man presented with a mass just behind the lower part of the left ear. A biopsy showed a moderately differentiated adenocarcinoma that was prostate-specific antigen (PSA)- and prostate-specific acid phosphatase (PSAP)-positive. This finding suggested a metastasis of a prostatic
Phosphatase enzymes. Cytochemical study of pleomorphic adenoma and normal human salivary glands.
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Human parotid glands, submandibular glands, and pleomorphic adenomas were examined by electron microscopic histochemistry. All epithelial cells of the normal salivary glands showed plasma membrane adenosine triphosphatase (ATPase) and inosine diphosphatase (IDPase) activity. However, myoepithelial
Loss of PTEN is associated with elevated EGFR and HER2 expression and worse prognosis in salivary gland cancer.
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BACKGROUND
Activity of the tumour-suppressor gene PTEN is reduced in different types of cancer and implicates non-responsiveness to targeted therapy. This study evaluates the gene and protein status of PTEN in salivary gland carcinomas.
METHODS
A total of 287 carcinomas of the major and minor
Predictors of cervical lymph node metastasis in salivary gland cancer.
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BACKGROUND
This study compares clinicopathological parameters with novel molecular markers for predicting cervical lymph node metastasis in salivary gland cancer.
METHODS
Three hundred sixteen salivary gland carcinomas were included in this study. Genomic epidermal growth factor receptor (EGFR),
Aberrations of MET are associated with copy number gain of EGFR and loss of PTEN and predict poor outcome in patients with salivary gland cancer.
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Hepatocyte growth factor receptor (MET) is a key driver of oncogenic transformation. Copy number gain and amplification of MET positively enhance tumour growth, invasiveness and metastasis in different cancer types. In the present study, 266 carcinomas of the major and minor salivary glands were
Localization of prostate-specific antigen-like immunoreactivity in human salivary gland and salivary gland tumors.
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Immunoreactivity of prostate-specific antigen (PSA), a kallikrein-like enzyme present in the seminal plasma, was demonstrated by indirect immunoperoxidase staining using a PSA antiserum in the apical cytoplasm along the luminal border of small-sized duct epithelial cells of the major salivary
Clinical activity of androgen deprivation therapy in patients with metastatic/relapsed androgen receptor-positive salivary gland cancers.
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BACKGROUND
Androgen deprivation therapy has some clinical activity in selected salivary gland cancer histotypes, with androgen receptor expression.
METHODS
We retrospectively analyzed patients with androgen receptor-expressing recurrent/metastatic salivary gland cancer, treated with androgen
Potential role for inhibition of protein phosphatase 2A tumor suppressor in salivary gland malignancies.
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The aetiology and pathogenesis of salivary gland malignancies remain unknown. To reveal novel molecular factors behind the development of salivary gland cancer, we performed gene expression analyses from Smgb-Tag mouse salivary gland samples. The overall purpose was to apply these results for
Isozyme phenotypes of polyoma virus tumors in mice.
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Isozyme profiles for 32 enzyme systems were studied in tumors induced by two strains of polyoma virus (2PTA and LID1), in two conventional mouse strains (C3H/BiDa and NIH), and in athymic (nude) mice of two genetic backgrounds (C3H/Hes nu/nu and NIH nu/nu). Tumors studied were: primary and
PTEN downregulates WD repeat‑containing protein 66 in salivary adenoid cystic carcinoma.
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Salivary adenoid cystic carcinoma (SACC) is one of the most common types of salivary gland cancer that causes substantial morbidity and mortality. Despite the substantial health burden of SACC, the molecular mechanisms underlying its development and progression remain poorly understood. We
Effect of glutathione and N-acetylcysteine on hepatocellular modifications induced by 2-acetylaminofluorene.
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The exposure of rats to a dietary regimen containing 2-acetylaminofluorene induces a sequence of hepatocellular alterations leading to the development of preneoplastic nodules. Groups of 2-acetylaminofluorene-treated rats were given glutathione or N-acetylcysteine to evaluate the effects of these
[Salivary gland myoepithelioma. Histological, histoenzymological and ultrastructural study].
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One case of myoepithelioma of the submandibular gland is reported. The tumor was composed of mixed spindle-shaped and plasmacytoid cells. The electron microscopy showed intracytoplasmic myofilaments, with variations in number and in repartition from one cell to another. Histoenzymologically, ATPasic
[Oncocyte cells in salivary tumors: frequency, histoenzymological and ultrastructural characteristics].
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Two types of salivary monomorphic adenomas, the so-called adenolymphoma and oncocytoma (75 cases in a series of 873 salivary gland tumors) were studied. These tumors were almost always located in major salivary glands (essentially in the parotid gland). They were much more common in men (85%) than
[Mucoepidermoid tumors of minor salivary glands. Clinical and pathologic correlations. Histoenzymologic and ultrastructural studies (author's transl)].
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In a series of 331 minor salivary gland tumors (malignant in about 55,3% of cases), mucoepidermoid tumors, after cystic adenoïd carcinomas, are the most frequent malignant tumors (21,5% of cases). They are much more common in women than in men. The average age of patients at presentation (52,2
Expression of PTEN, Androgen Receptor, HER2/neu, Cytokeratin 5/6, Estrogen Receptor-Beta, HMGA2, and PLAG1 in Salivary Duct Carcinoma.
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Salivary duct carcinoma (SDC) is an aggressive neoplasm that resembles high-grade invasive ductal carcinoma of the breast. It can develop de novo or from the malignant transformation of pleomorphic adenoma (PA). We performed immunohistochemical stains for phosphatase and tensin homologue [PTEN
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