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Strana 1 od 350 výsledky
Acute lysine supplementation does not improve hepatic or peripheral insulin sensitivity in older, overweight individuals.
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BACKGROUND
Lysine supplementation may have a positive influence on the regulation of glucose metabolism but it has not been tested in the geriatric population.
OBJECTIVE
We evaluated the impact of acute lysine supplementation using three randomized experimental scenarios: 1) oral glucose alone
The Nε-(carboxymethyl)lysine-RAGE axis: putative implications for the pathogenesis of obesity-related complications.
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Obesity is an important contributor to the burden of insulin resistance, Type 2 diabetes and cardiovascular disease. An important mechanism by which excess adiposity causes obesity-associated complications is the dysregulated production and secretion of biologically active molecules derived from
Protein utilization and lysine metabolism in obese and non-obese growing rats.
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Efficiency of protein utilization and lysine metabolism were studied in growing rats of the Zucker 13M strain, both obese and lean, and in the Charles River CD strain. When graded levels of wheat gluten or wheat gluten supplemented with lysine were fed to these three types of rats, no significant
Protein-Bound Plasma Nε-(Carboxymethyl)lysine Is Inversely Associated With Central Obesity and Inflammation and Significantly Explain a Part of the Central Obesity-Related Increase in Inflammation: The Hoorn and CODAM Studies.
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OBJECTIVE
Adipose tissue inflammation contributes to the development of complications, such as insulin resistance and type 2 diabetes mellitus. We previously reported that plasma levels of N(ε)-(carboxymethyl)lysine (CML) were decreased in obese subjects resulting from CML accumulation in adipose
Nε-(carboxymethyl)lysine-receptor for advanced glycation end product axis is a key modulator of obesity-induced dysregulation of adipokine expression and insulin resistance.
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OBJECTIVE
Dysregulation of inflammatory adipokines by the adipose tissue plays an important role in obesity-associated insulin resistance. Pathways leading to this dysregulation remain largely unknown. We hypothesized that the receptor for advanced glycation end products (RAGE) and the ligand
Lysine acetylation in obesity, diabetes and metabolic disease.
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Histone acetyltransferases (HATs) and histone deacetylases (HDACs) mediate acetylation and deacetylation of histone proteins and transcription factors. There is abundant evidence that these enzymes regulate the acetylation state of many cytoplasmic proteins, including lysine residues in important
Threonine, but Not Lysine and Methionine, Reduces Fat Accumulation by Regulating Lipid Metabolism in Obese Mice.
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Some amino acids (AAs) have been proven to suppress fat mass and improve insulin sensitivity. However, the impact of important essential AAs, threonine, lysine, and methionine, on obesity has not been clarified. In the present study, after an 8 week period of obesity induction, mice were grouped to
Obesity increases histone H3 lysine 9 and 18 acetylation at Tnfa and Ccl2 genes in mouse liver.
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Obesity contributes to the development of non-alcoholic fatty liver disease (NAFLD), which is characterized by the upregulated expression of two key inflammatory mediators: tumor necrosis factor (Tnfa) and monocyte chemotactic protein 1 (Mcp1; also known as Ccl2). However, the chromatin make-up at
Obesity-induced lysine acetylation increases cardiac fatty acid oxidation and impairs insulin signalling.
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OBJECTIVE
Lysine acetylation is a novel post-translational pathway that regulates the activities of enzymes involved in both fatty acid and glucose metabolism. We examined whether lysine acetylation controls heart glucose and fatty acid oxidation in high-fat diet (HFD) obese and SIRT3 knockout (KO)
[Study of diencephalo-hypophysial function in obesity, using the lysine-vasopressin test].
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[Evaluation of the function of the hypothalamus-hypophysis-adrenal gland system in obese children by means of the metopyrone and the lysine-8-vasopressin tests].
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[Effect of of L-dropa on the action of plasma cortisol during tests with lysine-8-vasopressin and insulin in essential obesity].
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[Effect of lysine vasopressin on plasma free fatty acid level in obese women].
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Epigenetic modification of the leptin promoter in diet-induced obese mice and the effects of N-3 polyunsaturated fatty acids.
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We report evidence of a detailed epigenetic modification of the leptin promoter and the effects of n-3 polyunsaturated fatty acids (n-3 PUFAs), which is closely associated with the leptin gene transcription in obesity. In the adipose tissue of diet induced obese (DIO) mice, methylation of the CpG
Characteristics and antioxidant activity of Maillard reaction products from psicose-lysine and fructose-lysine model systems.
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D-Psicose, an epimer of D-fructose isomerized at C-3 position, is a rare ketohexose that is thought to be beneficial for obese people and diabetic patients as a noncaloric sweetener. In the present study, model Maillard reaction products were obtained from D-psicose (or D-fructose) and L-lysine
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