meningococcal infections/opuch

Cerebrospinal fluid was tested for several enzymes (CPK, AST, GGTP, BP, BG, AK) in 97 patients with generalized meningococcal infection with prevalent CNS affliction. Marked changes were detected even in the absence of clinical signs of cerebral lesion that was typical only of meningococcemia.

[Clinical-pathogenetic features of meningococcal infection in children].

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Analysis features of clinical manifestation and pathogenetic mechanisms of development of meningococcal infection is introduced. It is emphasized that in most cases mentioned infection is characterized by mild course as nasopharyngitidis. However relatively seldom developing generalize form put this

[Diagnosis of cardiac lesions in the generalized form of meningococcal infection].

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Activity of the enzymes indicating heart affection in the acute period and in disease dynamics was increased in 97 patients with generalized forms of meningococcal infection. Signs of cytolysis, according to the data of enzymatic blood examination, were revealed to the utmost degree in patients with

[Clinical features of meningococcal infection in subjects with deficient terminal components of complement].

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OBJECTIVE To evaluate clinical characteristics of meningococcal disease (MD) in individuals with terminal complement component deficiency (TCCD) who are thousands times more susceptible to MD than complement-sufficient persons. METHODS 61 cases of MD in TCCD patients and 200 randomly selected cases

[Meningococcal disease: clinicopathological correlation].

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BACKGROUND Clinicopathological correlation studies of cases admitted as meningococcal disease are scarce, although they can serve to elucidate clinically obscure cases. METHODS A descriptive approach was used to analyze 42 necropsies following clinical diagnosis of meningococcal disease, verifying

Early cortical cytotoxic edema in meningococcal meningitis.

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Meningococcal disease frequently presents neurologic sequels via vascular, metabolic, or inflammatory processes. Understanding the underlying pathogenic mechanisms may influence both treatment and outcome. We present a 2-year-old child affected by Neisseria meningitidis sepsis, who on the second day

Protein C in the treatment of coagulopathy in meningococcal disease.

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OBJECTIVE To evaluate the clinical and laboratory effects of the substitution of protein C (PC) as an adjunct to conventional therapy in the treatment of purpura fulminans associated with meningococcal sepsis. METHODS case series. METHODS Medical and medical-surgical intensive care units of two

Meningococcal disease and meningitis.

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OBJECTIVE To review the literature relevant to diagnosis and management of meningococcal disease (MD). METHODS Non-systematic review of medical literature through the MEDLINE database using the terms meningococcal, septic shock, diagnosis, and treatment. Articles were selected according to their

[Guidelines for the diagnosis and treatment of meningococcal disease at hospitals].

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In Norway a hyperendemic situation has persisted since 1974 as regards meningococcal disease, with an adjusted annual incidence of almost 10 per 100,000 inhabitants. 80% of the cases are caused by group B meningococci, and the lethality has been about 10%. This article summarises the new Norwegian

[The enzyme diagnosis of liver involvement in meningococcal infection].

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A complex of blood enzymes (AST, ALT, AP, GTT) was studied in 84 patients with generalized forms of MI. The study showed that signs of liver involvement were noted in 15.4% of the patients on the 1st day of disease. Laboratory signs of cytolysis and cholestasis were revealed in most examinees on the

[Erythrocyte metabolism in meningococcal infection and purulent meningitis].

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The red blood cell metabolic parameters ATP, ADP, sigma AN, ATP/ADP, ATP/ATP, energy charge, PAD, 2,3-DPH, Pn were studied in 106 patients with generalized meningococcus infection (GMI) and meningitis of other etiology over their natural history. There was a typical adaptative red blood cell

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