nephrosclerosis/albumin

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Study of protein profiling of human urine in diabetic hypertensive nephropathy versus normal healthy controls.

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BACKGROUND This study describes the levels of protein acting as biomarkers affecting hypertensive diabetic nephropathy in the local Pakistani population. METHODS Patients with both hypertensive nephropathy and diabetes type 2 and normal healthy controls were recruited from Sheikh 380 Zayed Hospital,

ET(A) receptor antagonist ameliorates nephrosclerosis and left ventricular hypertrophy induced in rat by prolonged inhibition of nitric oxide synthesis.

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We investigated the ability of the ETA receptor antagonist T-0115 and the angiotensin-converting enzyme (ACE) inhibitor imidapril hydrochloride to prevent hypertensive complications induced in rats by chronic inhibition of nitric oxide (NO). Male Wistar rats were given distilled water (control),

Renal toxicity of albumin and other lipoproteins.

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Proteinuria may be an independent risk factor for nephrosclerosis. One potential mechanism has emerged with the finding that lipoproteins, including albumin, modulate the biology of mesangial cells and proximal tubular epithelial cells when internalized. Consequent alterations in renal-cell lipid

Clinical prognostic factors in biopsy-proven benign nephrosclerosis.

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BACKGROUND Hypertensive renal damage has become one of the most important causes of end-stage renal failure (ESRF) in Western countries. Affected patients rarely have a kidney biopsy and their diagnoses therefore remain uncertain. The objective of the present study was to examine patients suspected

[Hypertensive nephropathy: a growing cause of renal insufficiency].

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KIDNEY DISEASE AND HYPERTENSION: Hypertension is increasingly associated to renal insufficiency leading to end stage renal damage (ESRD), through several mechanisms involving renal vasculature. Ten to 25% of new ESRD patients are now categorized as hypertensive nephropathy and vascular renal

Renal-protective effect of nicousamide on hypertensive nephropathy in spontaneously hypertensive rats.

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Previous studies have demonstrated that a novel coumarin-aspirin derivative, nicousamide, has a significant renal-protective effect on diabetic nephropathy. The present study aimed to investigate the beneficial effect of nicousamide on hypertensive nephropathy, as well as the underlying mechanisms

Hypertensive nephropathy in children - do we diagnose early enough?

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OBJECTIVE The aim was to evaluate the level of neutrophil gelatinase-associated lipocalin (NGAL), interleukin 18 (IL-18) and retinol binding protein (RBP) in children with primary hypertension and no features of hypertensive nephropathy. METHODS The study group consisted of 19 children (15 males)

Hypertensive nephropathy and the gene for angiotensin-converting enzyme.

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To investigate the genetic determinants for microalbuminuria, we studied an insertion (I)/deletion (D) polymorphism of the angiotensin-converting enzyme (ACE) gene, which influences the plasma ACE level, in 333 consecutive hypertensive patients and 113 normotensive control subjects. The urinary

Antihypertensive agents prevent nephrosclerosis and left ventricular hypertrophy induced in rats by prolonged inhibition of nitric oxide synthesis.

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We investigated the ability of the angiotensin converting enzyme (ACE) inhibitor imidapril hydrochloride, and of the calcium channel blocker amlodipine besilate, to prevent nephrosclerosis and left ventricular hypertrophy (LVH) in rats with hypertension induced by chronic inhibition of nitric oxide

Changes in renal resistive index and urinary albumin excretion in hypertensive patients under long-term treatment with lisinopril or nifedipine GITS.

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BACKGROUND Increased renal vascular resistance and microalbuminuria are associated with hypertensive target organ damage and may be predictors of hypertensive nephrosclerosis. OBJECTIVE We investigated changes in renal resistive index (RI) and urinary albumin excretion (UAE) in a group of patients

Association of hypoalbuminemia with severe anemia in patients with diabetic nephrosclerosis.

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BACKGROUND Anemia in patients with early diabetes mellitus nephrosclerosis (DMN) is more severe than in patients with kidney disease of other origins, and the mechanism for this remains unclear. In this study, we carried out a retrospective study in order to identify the factors associated with

Relationship between body mass index and proteinuria in hypertensive nephrosclerosis: results from the African American Study of Kidney Disease and Hypertension (AASK) cohort.

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BACKGROUND Few studies have examined the association between obesity and markers of kidney injury in a chronic kidney disease population. We hypothesized that obesity is independently associated with proteinuria, a marker of chronic kidney disease progression. METHODS Observational cross-sectional

Effect of Proteinuria and Glomerular Filtration Rate on Renal Outcome in Patients with Biopsy-Proven Benign Nephrosclerosis.

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BACKGROUND Reduced estimated glomerular filtration rate (eGFR) and proteinuria are risk factors for end-stage renal disease (ESRD), of which benign nephrosclerosis is a common cause. However, few biopsy-based studies have assessed these associations. METHODS We performed retrospective cohort study

Larger nephron size, low nephron number, and nephrosclerosis on biopsy as predictors of kidney function after donating a kidney.

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It is unclear whether structural findings in the kidneys of living kidney donors predict postdonation kidney function. We studied living kidney donors who had a kidney biopsy during donation. Nephron size was measured by glomerular volume, cortex volume per glomerulus, and mean cross-sectional

Clinicopathological predictors for progression of chronic kidney disease in nephrosclerosis: a biopsy-based cohort study.

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UNASSIGNED Biopsy-based studies on nephrosclerosis are lacking and the clinicopathological predictors for progression of chronic kidney disease (CKD) are not well established. UNASSIGNED We retrospectively assessed 401 patients with biopsy-proven nephrosclerosis in Japan. Progression of CKD was

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