protein c deficiency/seizures

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Superior sagittal sinus thrombosis occurring at high altitude associated with protein C deficiency.

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A 42-year-old male presented with right-sided weakness, dysphasia and seizures while climbing the French Alps at an approximate altitude of 3,000 m. Imaging studies were consistent with superior sagittal sinus thrombosis with hemorrhage. Laboratory testing for thrombophilic states, 18 days after

[Cerebral sinus thrombosis in a young man with hereditary protein C deficiency].

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A 23-year-old man was admitted because of vomiting and severe, progressive headache. After admission, he suffered from a generalized clonic seizure, and developed right hemiparesis. Contrast-enhanced CT of the brain showed empty delta sign in the posterior part of the superior sagittal sinus and

Fetal hydrocephalus and neonatal stroke as the first presentation of protein C deficiency.

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Severe protein C-deficiency is a rare heritable thrombophilia of the newborn. Infants with biallelic PROC mutations present purpura fulminans and intracranial thromboembolism, while the prenatal onset of mutated heterozygotes remains unclear. We herewith present the first case of fetal

A case of protein C deficiency associated with cerebral infarction and obstruction of deep leg and inferior mesenteric veins.

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Protein C, a vitamin K-dependent protein, is a blood coagulation inhibitor. Its deficiency causes systemic thrombosis. A 31-year-old woman developed cerebral infarction followed by late psychomotor seizures, and thrombosis in the inferior mesenteric vein and bilateral crural veins. Her parents were

[Protein C deficiency and cerebral venous thrombosis in pregnancy].

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A fatal case of cerebral venous thrombosis associated with protein C deficiency is reported. It occurred at six months of gestation in a 25 year old patient during her first pregnancy. She had generalized seizures. Computed axial tomography displayed cerebral haemorrhagic infarction, and the carotid

[Thyroid crisis and protein C deficiency in a case of superior sagittal sinus thrombosis].

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We report the case of a 28-year-old woman who presented simultaneously with superior sagittal sinus thrombosis and thyroid crisis, and was subsequently found to have protein C deficiency. February 3, 2003, she admitted complaining of abdominal pain. The diagnosis of appendicitis was made, and she

[A Case Report of Congenital Protein C Deficiency with Cerebral Venous Sinus Thrombosis during Early Pregnancy Period].

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A 35-year-old woman at eight weeks of gestation in her second pregnancy presented with generalized seizures. Magnetic resonance images revealed a small hemorrhagic infarction in the left frontal lobe, and magnetic resonance venography indicated cerebral venous sinus thrombosis. After

Psychiatric remission with warfarin: Should psychosis be addressed as plasminogen activator imbalance?

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BACKGROUND Psychotic patients are at increased risk of thromboembolism that cannot be ascribed to physical restraint or medication. Patients with chronic schizophrenia or long-term depressive illness do not display ischemic brain injuries on magnetic resonance imaging, as expected in patients with

[Status of hereditary hypercoagulability and cerebral venous thrombosis. Report of 3 cases].

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Hereditary hypercoagulability has been identified as risk factor in approximately 30% of cerebral venous thrombosis cases. We report three females with this association. A 38 years old female with a history of deep venous thrombosis of the lower limb, presented with headache, vomiting and a

Subcortical infarction in children.

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OBJECTIVE This report examines the occurrence of subcortical infarction in 5 children, reviews the English literature, and discusses evaluation of this uncommon childhood illness. METHODS Clinical characteristics and neurological follow-up were examined in children who presented with subcortical

Neonatal deep white matter venous infarction and liquefaction: a pseudo-abscess lesion.

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BACKGROUND Deep white matter hemorrhagic venous infarction with subsequent cavitation due to necrosis and liquefaction has been described in neonates and may be associated with infection and meningitis. In our experience, the MRI pattern of these lesions is confused with the pattern seen with

[Cerebral venous thrombosis: when etiology makes the difference].

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OBJECTIVE To make a retrospective study of the clinical, etiological, diagnostic and prognostic features of cerebral vein and sinus thrombosis (CVST) diagnosed at our University Hospital. METHODS We performed a systematic research of the clinical records of our University Hospital's electronic

Etiological profile of stroke and its relation with prothrombotic states.

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OBJECTIVE To study the etiological profile of childhood stroke and its relation with prothrombotic states. METHODS Children with acute stroke with no evidence of CNS infection or head injury were studied. Stroke was confirmed by CT scan and further evaluated by MRI. Cardiac status was assessed with

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