Stran 1 iz 249 rezultatov
OBJECTIVE
To study the hypothesis that environmental tobacco smoke exposure in infants diagnosed with an apparent life-threatening event (ALTE) could be a contributing factor to gastroesophageal reflux.
METHODS
Thirty-four polygraphic recordings of combined esophageal pH study and respiration in
OBJECTIVE
To investigate the effect of smokeless tobacco (ST), cigar and/or pipe smoking (CP) on the development of Barrett esophagus (BE) in white male patients with gastroesophageal reflux disease (GERD).
METHODS
A total of 1015 records of white male adults with BE (cases; n=508) or GERD
Transdermal nicotine delivery systems are widely used in smoking cessation. The purpose of this study was to determine whether common symptoms of pyrosis and dyspepsia associated with these patches are related to gastroesophageal reflux or esophageal dysmotility. Twenty-seven paid volunteer
OBJECTIVE
Studies have shown that cigarette smoking acutely increases gastroesophageal reflux (GER). Patients prescribed transdermal nicotine often complain of pyrosis. The purpose of this study was to ascertain the effect of transdermal nicotine on GER.
METHODS
Twenty volunteers (12 smokers and
Gastro-oesophageal reflux disease (GORD) develops when reflux of gastric content causes troublesome symptoms or complications. The main symptoms are heartburn and acid regurgitation and complications include oesophagitis, strictures, Barrett's oesophagus and oesophageal adenocarcinoma. In addition
OBJECTIVE
Tobacco smoking increases the risk of gastroesophageal reflux symptoms (GERS), but whether tobacco smoking cessation improves GERS is unclear. The aim of this study was to clarify if tobacco smoking cessation improves GERS.
METHODS
The study was based on the Nord-Trøndelag health study
BACKGROUND
Dental erosion (DE), one of oral hard tissue diseases, is one of the extraoesophageal symptoms defined as the Montreal Definition and Classification of gastroesophageal reflux disease (GERD). However, no study evaluated the relationship between GERD and oral soft tissues. We hypothesized
The first step in the management of gastroesophageal reflux disease (GERD) is lifestyle modification, with dietary recommendations such as increasing fiber intake and lowering dietary fat. While there is some physiological evidence that various foods as well as alcohol and tobacco affect the
OBJECTIVE
We conducted a nested case-control study to examine the prevalence and risk factors for current gastroesophageal reflux disease (GERD) symptoms in young adults with a history of childhood GERD.
METHODS
We identified a cohort of individuals diagnosed with GERD in childhood during 1995-1996,
To investigate the potentially bidirectional association between gastroesophageal reflux symptoms (GERS) and sleep disturbances/ insomnia disorders.
We assessed the incidence of new-onset of self-reported GERS, sleep disturbances, and insomnia disorders in a population-based longitudinal cohort
OBJECTIVE
To evaluate the efficacy of a specific protocol of conservative therapy for infant gastroesophageal reflux.
METHODS
Retrospective evaluation of the response to telephone teaching of conservative therapy by a single instructor as part of the screening process for a pharmacotherapy study of
OBJECTIVE
To identify infant health outcomes that have been empirically associated with environmental tobacco smoke (ETS) exposure in current international research literature.
METHODS
Reports of infant ETS exposure, obtained using computerized searches of CINAHL and Medline databases, were
BACKGROUND
Environmental tobacco smoke (ETS) negatively affects children with asthma. The prevalence of ETS exposure among children with poor asthma control may be changing. Importantly, the mechanisms by which ETS worsens asthma control are poorly understood.
OBJECTIVE
We describe how ETS affects
Introduction: Tobacco use worsens gastrointestinal reflux disease (GERD). Smoking cessation improves GERD symptoms, but its impact on the efficacy of laparoscopic anti-reflux surgery (LARS) is unclear. In this retrospective cohort study,
The role of smoking in the pathogenesis of gastrooesophageal reflux disease has been controversial since the early 1970s when Stanciu reported the two to be 92% epidemiologically associated (a study subsequently challenged by inconsistencies in the observational data). Mechanistically, reflux